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10.1016/j.yjmcc.2015.11.015 http://scihub22266oqcxt.onion/10.1016/j.yjmcc.2015.11.015 C4846552!4846552 !26593722     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26593722 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Mol+Cell+Cardiol 2016 ; 93 (ä): 149-55 Nephropedia Template TP {{tp|p=26593722 |t=2016. Monocyte and macrophage contributions to cardiac remodeling |pdf=|usr=}}{{26593722 }} gab.com Text Monocyte and macrophage contributions to cardiac remodeling JO: J Mol Cell Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26593722 #FOAvip The mammalian heart contains a population of resident macrophages that expands in response to myocardial infarction and hemodynamic stress. This expansion occurs likely through both local macrophage proliferation and monocyte recruitment. Given the role of macrophages in tissue remodeling, their contribution to adaptive processes in the heart is conceivable but currently poorly understood. In this review, we discuss monocyte and macrophage heterogeneity associated with cardiac stress, the cell's potential contribution to the pathogenesis of cardiac fibrosis, and describe different tools to study and characterize these innate immune cells. Finally, we highlight their potential role as therapeutic targets. Twit Text FOAVip Monocyte and macrophage contributions to cardiac remodeling ????J Mol Cell Cardiol http://www.kidney.de/pget.php?&tw=1&pmid=26593722 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26593722 #FOAvip English Wikipedia <ref>{{Cite pmid|26593722 }}</ref> Monocyte and macrophage contributions to cardiac remodeling #MMPMID26593722 Hulsmans M ; Sam F ; Nahrendorf M J Mol Cell Cardiol 2016[Apr]; 93 (ä): 149-55 PMID26593722 show ga The mammalian heart contains a population of resident macrophages that expands in response to myocardial infarction and hemodynamic stress. This expansion occurs likely through both local macrophage proliferation and monocyte recruitment. Given the role of macrophages in tissue remodeling, their contribution to adaptive processes in the heart is conceivable but currently poorly understood. In this review, we discuss monocyte and macrophage heterogeneity associated with cardiac stress, the cell's potential contribution to the pathogenesis of cardiac fibrosis, and describe different tools to study and characterize these innate immune cells. Finally, we highlight their potential role as therapeutic targets. |*Ventricular Remodeling [MESH] |Animals [MESH] |Biomarkers [MESH] |Cell Communication [MESH] |Cell Proliferation [MESH] |Fibrosis [MESH] |Humans [MESH] |Macrophages/*immunology/*metabolism [MESH] |Monocytes/*immunology/*metabolism [MESH] |Myocardium/metabolism/pathology [MESH] |Phenotype [MESH] |Signal Transduction [MESH]Monocyte and macrophage contributions to cardiac remodeling (epmc).pdf DeepDyve Pubget Overpricing