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10.3109/07853890.2014.949299 http://scihub22266oqcxt.onion/10.3109/07853890.2014.949299 C4438354!4438354 !25117779     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25117779 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Ann+Med 2014 ; 46 (8 ): 597-606 Nephropedia Template TP {{tp|p=25117779 |t=2014. Molecular basis and genetic predisposition to intracranial aneurysm |pdf=|usr=}}{{25117779 }} gab.com Text Molecular basis and genetic predisposition to intracranial aneurysm JO: Ann Med http://www.kidney.de/pget.php?&tw=1&pmid=25117779 #FOAvip Intracranial aneurysms, also called cerebral aneurysms, are dilatations in the arteries that supply blood to the brain. Rupture of an intracranial aneurysm leads to a subarachnoid hemorrhage, which is fatal in about 50% of the cases. Intracranial aneurysms can be repaired surgically or endovascularly, or by combining these two treatment modalities. They are relatively common with an estimated prevalence of unruptured aneurysms of 2%-6% in the adult population, and are considered a complex disease with both genetic and environmental risk factors. Known risk factors include smoking, hypertension, increasing age, and positive family history for intracranial aneurysms. Identifying the molecular mechanisms underlying the pathogenesis of intracranial aneurysms is complex. Genome-wide approaches such as DNA linkage and genetic association studies, as well as microarray-based mRNA expression studies, provide unbiased approaches to identify genetic risk factors and dissecting the molecular pathobiology of intracranial aneurysms. The ultimate goal of these studies is to use the information in clinical practice to predict an individual's risk for developing an aneurysm or monitor its growth or rupture risk. Another important goal is to design new therapies based on the information on mechanisms of disease processes to prevent the development or halt the progression of intracranial aneurysms. Twit Text FOAVip Molecular basis and genetic predisposition to intracranial aneurysm ????Ann Med http://www.kidney.de/pget.php?&tw=1&pmid=25117779 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25117779 #FOAvip English Wikipedia <ref>{{Cite pmid|25117779 }}</ref> Molecular basis and genetic predisposition to intracranial aneurysm #MMPMID25117779 Tromp G ; Weinsheimer S ; Ronkainen A ; Kuivaniemi H Ann Med 2014[Dec]; 46 (8 ): 597-606 PMID25117779 show ga Intracranial aneurysms, also called cerebral aneurysms, are dilatations in the arteries that supply blood to the brain. Rupture of an intracranial aneurysm leads to a subarachnoid hemorrhage, which is fatal in about 50% of the cases. Intracranial aneurysms can be repaired surgically or endovascularly, or by combining these two treatment modalities. They are relatively common with an estimated prevalence of unruptured aneurysms of 2%-6% in the adult population, and are considered a complex disease with both genetic and environmental risk factors. Known risk factors include smoking, hypertension, increasing age, and positive family history for intracranial aneurysms. Identifying the molecular mechanisms underlying the pathogenesis of intracranial aneurysms is complex. Genome-wide approaches such as DNA linkage and genetic association studies, as well as microarray-based mRNA expression studies, provide unbiased approaches to identify genetic risk factors and dissecting the molecular pathobiology of intracranial aneurysms. The ultimate goal of these studies is to use the information in clinical practice to predict an individual's risk for developing an aneurysm or monitor its growth or rupture risk. Another important goal is to design new therapies based on the information on mechanisms of disease processes to prevent the development or halt the progression of intracranial aneurysms. |*Genetic Predisposition to Disease [MESH] |*Genome-Wide Association Study [MESH] |Gene Expression [MESH] |Gene-Environment Interaction [MESH] |Humans [MESH] |Intracranial Aneurysm/*genetics [MESH] |Oligonucleotide Array Sequence Analysis [MESH] |Polymorphism, Single Nucleotide [MESH]Molecular basis and genetic predisposition to intracranial aneurysm (epmc).pdf DeepDyve Pubget Overpricing