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10.1155/2017/7803029 http://scihub22266oqcxt.onion/10.1155/2017/7803029 C5303843!5303843 !28251158     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28251158 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Biomed+Res+Int 2017 ; 2017 (ä): 7803029 Nephropedia Template TP {{tp|p=28251158 |t=2017. Molecular and Histopathological Changes Associated with Keratoconus |pdf=|usr=}}{{28251158 }} gab.com Text Molecular and Histopathological Changes Associated with Keratoconus JO: Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=28251158 #FOAvip Keratoconus (KC) is a corneal thinning disorder that leads to loss of visual acuity through ectasia, opacity, and irregular astigmatism. It is one of the leading indicators for corneal transplantation in the Western countries. KC usually starts at puberty and progresses until the third or fourth decade; however its progression differs among patients. In the keratoconic cornea, all layers except the endothelium have been shown to have histopathological structural changes. Despite numerous studies in the last several decades, the mechanisms of KC development and progression remain unclear. Both genetic and environmental factors may contribute to the pathogenesis of KC. Many previous articles have reviewed the genetic aspects of KC, but in this review we summarize the histopathological features of different layers of cornea and discuss the differentially expressed proteins in the KC-affected cornea. This summary will help emphasize the major molecular defects in KC and identify additional research areas related to KC, potentially opening up possibilities for novel methods of KC prevention and therapeutic intervention. Twit Text FOAVip Molecular and Histopathological Changes Associated with Keratoconus ????Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=28251158 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28251158 #FOAvip English Wikipedia <ref>{{Cite pmid|28251158 }}</ref> Molecular and Histopathological Changes Associated with Keratoconus #MMPMID28251158 Khaled ML ; Helwa I ; Drewry M ; Seremwe M ; Estes A ; Liu Y Biomed Res Int 2017[]; 2017 (ä): 7803029 PMID28251158 show ga Keratoconus (KC) is a corneal thinning disorder that leads to loss of visual acuity through ectasia, opacity, and irregular astigmatism. It is one of the leading indicators for corneal transplantation in the Western countries. KC usually starts at puberty and progresses until the third or fourth decade; however its progression differs among patients. In the keratoconic cornea, all layers except the endothelium have been shown to have histopathological structural changes. Despite numerous studies in the last several decades, the mechanisms of KC development and progression remain unclear. Both genetic and environmental factors may contribute to the pathogenesis of KC. Many previous articles have reviewed the genetic aspects of KC, but in this review we summarize the histopathological features of different layers of cornea and discuss the differentially expressed proteins in the KC-affected cornea. This summary will help emphasize the major molecular defects in KC and identify additional research areas related to KC, potentially opening up possibilities for novel methods of KC prevention and therapeutic intervention. |Cornea/pathology [MESH] |Homeostasis [MESH] |Hormones/metabolism [MESH] |Humans [MESH] |Keratoconus/*genetics/*pathology [MESH]Molecular and Histopathological Changes Associated with Keratoconus (epmc).pdf DeepDyve Pubget Overpricing