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10.2174/1573404813666170306163448 http://scihub22266oqcxt.onion/10.2174/1573404813666170306163448 C5925869!5925869 !29861704     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29861704 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Womens+Health+Rev 2018 ; 14 (2 ): 106-116 Nephropedia Template TP {{tp|p=29861704 |t=2018. Molecular and Cellular Pathogenesis of Endometriosis |pdf=|usr=}}{{29861704 }} gab.com Text Molecular and Cellular Pathogenesis of Endometriosis JO: Curr Womens Health Rev http://www.kidney.de/pget.php?&tw=1&pmid=29861704 #FOAvip BACKGROUND: A substantial body of studies supports the view that molecular and cellular features of endometriotic lesions differ from those of eutopic endometrium. Apart from that, evidence exists that the eutopic endometrium from pa-tients with endometriosis differs from that of females without endometriosis. OBJECTIVE: Aberrant expression profiles include a number of non-steroid signaling pathways that exert their putative influ-ence on the pathogenesis of endometriosis at least in part via crosstalk(s) with estrogen-mediated mechanisms. A rational to focus research on non-steroid signal pathways is that they might be remunerative targets for the development and selection of novel therapeutics to treat endometriosis possibly without affecting estrogen levels. RESULTS AND CONCLUSION: In this article, we describe molecular and cellular features of endometriotic lesions and focus on the canonical WNT/?-signaling pathway, a key regulatory system in biology (including stem cell homeostasis) and often in pathophysiological conditions such as endometriosis. Recently emerged novel biological concepts in signal transduction and gene regulation like exosomes and microRNAs are discussed in their putative role in the pathogenesis of endometriosis. Twit Text FOAVip Molecular and Cellular Pathogenesis of Endometriosis ????Curr Womens Health Rev http://www.kidney.de/pget.php?&tw=1&pmid=29861704 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29861704 #FOAvip English Wikipedia <ref>{{Cite pmid|29861704 }}</ref> Molecular and Cellular Pathogenesis of Endometriosis #MMPMID29861704 Klemmt PAB ; Starzinski-Powitz A Curr Womens Health Rev 2018[Jun]; 14 (2 ): 106-116 PMID29861704 show ga BACKGROUND: A substantial body of studies supports the view that molecular and cellular features of endometriotic lesions differ from those of eutopic endometrium. Apart from that, evidence exists that the eutopic endometrium from pa-tients with endometriosis differs from that of females without endometriosis. OBJECTIVE: Aberrant expression profiles include a number of non-steroid signaling pathways that exert their putative influ-ence on the pathogenesis of endometriosis at least in part via crosstalk(s) with estrogen-mediated mechanisms. A rational to focus research on non-steroid signal pathways is that they might be remunerative targets for the development and selection of novel therapeutics to treat endometriosis possibly without affecting estrogen levels. RESULTS AND CONCLUSION: In this article, we describe molecular and cellular features of endometriotic lesions and focus on the canonical WNT/?-signaling pathway, a key regulatory system in biology (including stem cell homeostasis) and often in pathophysiological conditions such as endometriosis. Recently emerged novel biological concepts in signal transduction and gene regulation like exosomes and microRNAs are discussed in their putative role in the pathogenesis of endometriosis. äMolecular and Cellular Pathogenesis of Endometriosis (epmc).pdf DeepDyve Pubget Overpricing