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10.3389/fonc.2017.00028 http://scihub22266oqcxt.onion/10.3389/fonc.2017.00028 C5334363!5334363 !28316954     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28316954 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28316954 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Oncol 2017 ; 7 (ä): 28 Nephropedia Template TP {{tp|p=28316954 |t=2017. Molecular Pathways Controlling Autophagy in Pancreatic Cancer |pdf=|usr=}}{{28316954 }} gab.com Text Molecular Pathways Controlling Autophagy in Pancreatic Cancer JO: Front Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28316954 #FOAvip Pancreatic ductal adenocarcinoma (PDAC) is one of the few cancer types where the 5-year survival rate shows no improvement. Despite conflicting evidence, the majority of data points to an essential role for autophagy in PDAC growth and survival, in particular constitutively activated autophagy, can provide crucial fuel to PDAC tumor cells in their nutrient-deprived environment. Autophagy, which is required for cell homeostasis, can both suppress and promote tumorigenesis and tumor survival in a context-dependent manner. Protein by protein, the mystery of how PDAC abuses the cell's homeostasis system for its malignant growth has recently begun to be unraveled. In this review, we focus on how autophagy is responsible for growth and development of PDAC tumors and where autophagy and the mechanisms controlling it fit into PDAC metabolism. Understanding the range of pathways controlling autophagy and their interplay in PDAC could open the way for new therapeutic avenues. Twit Text FOAVip Molecular Pathways Controlling Autophagy in Pancreatic Cancer ????Front Oncol http://www.kidney.de/pget.php?&tw=1&pmid=28316954 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28316954 #FOAvip English Wikipedia <ref>{{Cite pmid|28316954 }}</ref> Molecular Pathways Controlling Autophagy in Pancreatic Cancer #MMPMID28316954 New M ; Van Acker T ; Long JS ; Sakamaki JI ; Ryan KM ; Tooze SA Front Oncol 2017[]; 7 (ä): 28 PMID28316954 show ga Pancreatic ductal adenocarcinoma (PDAC) is one of the few cancer types where the 5-year survival rate shows no improvement. Despite conflicting evidence, the majority of data points to an essential role for autophagy in PDAC growth and survival, in particular constitutively activated autophagy, can provide crucial fuel to PDAC tumor cells in their nutrient-deprived environment. Autophagy, which is required for cell homeostasis, can both suppress and promote tumorigenesis and tumor survival in a context-dependent manner. Protein by protein, the mystery of how PDAC abuses the cell's homeostasis system for its malignant growth has recently begun to be unraveled. In this review, we focus on how autophagy is responsible for growth and development of PDAC tumors and where autophagy and the mechanisms controlling it fit into PDAC metabolism. Understanding the range of pathways controlling autophagy and their interplay in PDAC could open the way for new therapeutic avenues. äMolecular Pathways Controlling Autophagy in Pancreatic Cancer (epmc).pdf DeepDyve Pubget Overpricing