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10.1038/ncomms15422 http://scihub22266oqcxt.onion/10.1038/ncomms15422 C5458507!5458507 !28541307     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28541307 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Nat+Commun 2017 ; 8 (ä): 15422 Nephropedia Template TP {{tp|p=28541307 |t=2017. Modeling the process of human tumorigenesis |pdf=|usr=}}{{28541307 }} gab.com Text Modeling the process of human tumorigenesis JO: Nat Commun http://www.kidney.de/pget.php?&tw=1&pmid=28541307 #FOAvip Modelling the genesis of human cancers is at a scientific turning point. Starting from primary sources of normal human cells, it is now possible to reproducibly generate several types of malignant cell populations. Powerful methods for clonally tracking and manipulating their appearance and progression in serially transplanted immunodeficient mice are also in place. These developments circumvent historic drawbacks inherent in analyses of cancers produced in model organisms, established human malignant cell lines, or highly heterogeneous patient samples. In this review, we survey the advantages, contributions and limitations of current de novo human tumorigenesis strategies and note several exciting prospects on the horizon. Twit Text FOAVip Modeling the process of human tumorigenesis ????Nat Commun http://www.kidney.de/pget.php?&tw=1&pmid=28541307 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28541307 #FOAvip English Wikipedia <ref>{{Cite pmid|28541307 }}</ref> Modeling the process of human tumorigenesis #MMPMID28541307 Balani S ; Nguyen LV ; Eaves CJ Nat Commun 2017[May]; 8 (ä): 15422 PMID28541307 show ga Modelling the genesis of human cancers is at a scientific turning point. Starting from primary sources of normal human cells, it is now possible to reproducibly generate several types of malignant cell populations. Powerful methods for clonally tracking and manipulating their appearance and progression in serially transplanted immunodeficient mice are also in place. These developments circumvent historic drawbacks inherent in analyses of cancers produced in model organisms, established human malignant cell lines, or highly heterogeneous patient samples. In this review, we survey the advantages, contributions and limitations of current de novo human tumorigenesis strategies and note several exciting prospects on the horizon. |*Carcinogenesis/genetics [MESH] |*Models, Biological [MESH] |Animals [MESH] |CRISPR-Cas Systems [MESH] |Cell Transformation, Neoplastic [MESH] |Evolution, Molecular [MESH] |Humans [MESH] |Induced Pluripotent Stem Cells/pathology [MESH] |Mice [MESH] |Mice, Transgenic [MESH] |Mutation [MESH] |Neoplasms, Experimental/etiology [MESH] |Neoplasms/etiology/genetics/pathology [MESH] |Neoplastic Stem Cells/pathology [MESH]Modeling the process of human tumorigenesis (epmc).pdf DeepDyve Pubget Overpricing