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10.3390/cells7030018 http://scihub22266oqcxt.onion/10.3390/cells7030018 C5870350!5870350 !29510535     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29510535 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29510535 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Cells 2018 ; 7 (3 ): ä Nephropedia Template TP {{tp|p=29510535 |t=2018. Miro-Working beyond Mitochondria and Microtubules |pdf=|usr=}}{{29510535 }} gab.com Text Miro-Working beyond Mitochondria and Microtubules JO: Cells http://www.kidney.de/pget.php?&tw=1&pmid=29510535 #FOAvip The small GTPase Miro is best known for its regulation of mitochondrial movement by engaging with the microtubule-based motor proteins kinesin and dynein. Very recent findings have now showed that Miro also targets peroxisomes and regulates microtubule-dependent peroxisome motility. Moreover, Miro recruits and stabilizes the myosin motor Myo19 at the mitochondria to enable actin-based mitochondria movement, which is important for mitochondrial segregation during mitosis. Miro thus has much broader functions that previously known, and these new findings may have important implications on disease pathology. Twit Text FOAVip Miro-Working beyond Mitochondria and Microtubules ????Cells http://www.kidney.de/pget.php?&tw=1&pmid=29510535 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29510535 #FOAvip English Wikipedia <ref>{{Cite pmid|29510535 }}</ref> Miro-Working beyond Mitochondria and Microtubules #MMPMID29510535 Tang BL Cells 2018[Mar]; 7 (3 ): ä PMID29510535 show ga The small GTPase Miro is best known for its regulation of mitochondrial movement by engaging with the microtubule-based motor proteins kinesin and dynein. Very recent findings have now showed that Miro also targets peroxisomes and regulates microtubule-dependent peroxisome motility. Moreover, Miro recruits and stabilizes the myosin motor Myo19 at the mitochondria to enable actin-based mitochondria movement, which is important for mitochondrial segregation during mitosis. Miro thus has much broader functions that previously known, and these new findings may have important implications on disease pathology. äMiro-Working beyond Mitochondria and Microtubules (epmc).pdf DeepDyve Pubget Overpricing