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10.1016/j.reprotox.2016.10.001 http://scihub22266oqcxt.onion/10.1016/j.reprotox.2016.10.001 C5365353!5365353 !27760374     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27760374 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27760374 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Reprod+Toxicol 2017 ; 68 (ä): 3-33 Nephropedia Template TP {{tp|p=27760374 |t=2017. Metabolism disrupting chemicals and metabolic disorders |pdf=|usr=}}{{27760374 }} gab.com Text Metabolism disrupting chemicals and metabolic disorders JO: Reprod Toxicol http://www.kidney.de/pget.php?&tw=1&pmid=27760374 #FOAvip The recent epidemics of metabolic diseases, obesity, type 2 diabetes(T2D), liver lipid disorders and metabolic syndrome have largely been attributed to genetic background and changes in diet, exercise and aging. However, there is now considerable evidence that other environmental factors may contribute to the rapid increase in the incidence of these metabolic diseases. This review will examine changes to the incidence of obesity, T2D and non-alcoholic fatty liver disease (NAFLD), the contribution of genetics to these disorders and describe the role of the endocrine system in these metabolic disorders. It will then specifically focus on the role of endocrine disrupting chemicals (EDCs) in the etiology of obesity, T2D and NAFLD while finally integrating the information on EDCs on multiple metabolic disorders that could lead to metabolic syndrome. We will specifically examine evidence linking EDC exposures during critical periods of development with metabolic diseases that manifest later in life and across generations. Twit Text FOAVip Metabolism disrupting chemicals and metabolic disorders ????Reprod Toxicol http://www.kidney.de/pget.php?&tw=1&pmid=27760374 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27760374 #FOAvip English Wikipedia <ref>{{Cite pmid|27760374 }}</ref> Metabolism disrupting chemicals and metabolic disorders #MMPMID27760374 Heindel JJ ; Blumberg B ; Cave M ; Machtinger R ; Mantovani A ; Mendez MA ; Nadal A ; Palanza P ; Panzica G ; Sargis R ; Vandenberg LN ; Vom Saal F Reprod Toxicol 2017[Mar]; 68 (ä): 3-33 PMID27760374 show ga The recent epidemics of metabolic diseases, obesity, type 2 diabetes(T2D), liver lipid disorders and metabolic syndrome have largely been attributed to genetic background and changes in diet, exercise and aging. However, there is now considerable evidence that other environmental factors may contribute to the rapid increase in the incidence of these metabolic diseases. This review will examine changes to the incidence of obesity, T2D and non-alcoholic fatty liver disease (NAFLD), the contribution of genetics to these disorders and describe the role of the endocrine system in these metabolic disorders. It will then specifically focus on the role of endocrine disrupting chemicals (EDCs) in the etiology of obesity, T2D and NAFLD while finally integrating the information on EDCs on multiple metabolic disorders that could lead to metabolic syndrome. We will specifically examine evidence linking EDC exposures during critical periods of development with metabolic diseases that manifest later in life and across generations. |Animals [MESH] |Endocrine Disruptors/*toxicity [MESH] |Female [MESH] |Humans [MESH] |Metabolic Diseases/*chemically induced/epidemiology/genetics [MESH] |Metabolic Networks and Pathways/*drug effects/genetics [MESH] |Neurosecretory Systems/*drug effects/embryology [MESH] |Pregnancy [MESH] |Prenatal Exposure Delayed Effects/epidemiology/*etiology/genetics [MESH]Metabolism disrupting chemicals and metabolic disorders (epmc).pdf DeepDyve Pubget Overpricing