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10.1016/j.jacbts.2016.11.009 http://scihub22266oqcxt.onion/10.1016/j.jacbts.2016.11.009 C5609457!5609457 !28944310     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28944310 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       JACC+Basic+Transl+Sci 2017 ; 2 (3 ): 297-310 Nephropedia Template TP {{tp|p=28944310 |t=2017. Metabolic Origins of Heart Failure |pdf=|usr=}}{{28944310 }} gab.com Text Metabolic Origins of Heart Failure JO: JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=28944310 #FOAvip For more than half a century, metabolic perturbations have been explored in the failing myocardium, highlighting a reversion to a more fetal-like metabolic profile (characterized by depressed fatty acid oxidation and concomitant increased reliance on glucose utilization). More recently, alterations in ketone body and amino acid/protein metabolism have been described during heart failure, as well as mitochondrial dysfunction and perturbed metabolic signaling (e.g., acetylation, O-GlcNAcylation). Although numerous mechanisms are likely involved, the current review provides recent advances regarding the metabolic origins of heart failure, and their potential contribution toward contractile dysfunction of the heart. Twit Text FOAVip Metabolic Origins of Heart Failure ????JACC Basic Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=28944310 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28944310 #FOAvip English Wikipedia <ref>{{Cite pmid|28944310 }}</ref> Metabolic Origins of Heart Failure #MMPMID28944310 Wende AR ; Brahma MK ; McGinnis GR ; Young ME JACC Basic Transl Sci 2017[Jun]; 2 (3 ): 297-310 PMID28944310 show ga For more than half a century, metabolic perturbations have been explored in the failing myocardium, highlighting a reversion to a more fetal-like metabolic profile (characterized by depressed fatty acid oxidation and concomitant increased reliance on glucose utilization). More recently, alterations in ketone body and amino acid/protein metabolism have been described during heart failure, as well as mitochondrial dysfunction and perturbed metabolic signaling (e.g., acetylation, O-GlcNAcylation). Although numerous mechanisms are likely involved, the current review provides recent advances regarding the metabolic origins of heart failure, and their potential contribution toward contractile dysfunction of the heart. äMetabolic Origins of Heart Failure (epmc).pdf DeepDyve Pubget Overpricing