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10.1038/nrc.2016.40 http://scihub22266oqcxt.onion/10.1038/nrc.2016.40 C5336345!5336345 !27220482     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27220482 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Nat+Rev+Cancer 2016 ; 16 (6 ): 387-98 Nephropedia Template TP {{tp|p=27220482 |t=2016. Mechanisms of human lymphoid chromosomal translocations |pdf=|usr=}}{{27220482 }} gab.com Text Mechanisms of human lymphoid chromosomal translocations JO: Nat Rev Cancer http://www.kidney.de/pget.php?&tw=1&pmid=27220482 #FOAvip Analysis of chromosomal translocation sequence locations in human lymphomas has provided valuable clues about the mechanism of the translocations and when they occur. Biochemical analyses on the mechanisms of DNA breakage and rejoining permit formulation of detailed models of the human chromosomal translocation process in lymphoid neoplasms. Most human lymphomas are derived from B cells in which a DNA break at an oncogene is initiated by activation-induced deaminase (AID). The partner locus in many cases is located at one of the antigen receptor loci, and this break is generated by the recombination activating gene (RAG) complex or by AID. After breakage, the joining process typically occurs by non-homologous DNA end-joining (NHEJ). Some of the insights into this mechanism also apply to translocations that occur in non-lymphoid neoplasms. Twit Text FOAVip Mechanisms of human lymphoid chromosomal translocations ????Nat Rev Cancer http://www.kidney.de/pget.php?&tw=1&pmid=27220482 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27220482 #FOAvip English Wikipedia <ref>{{Cite pmid|27220482 }}</ref> Mechanisms of human lymphoid chromosomal translocations #MMPMID27220482 Lieber MR Nat Rev Cancer 2016[May]; 16 (6 ): 387-98 PMID27220482 show ga Analysis of chromosomal translocation sequence locations in human lymphomas has provided valuable clues about the mechanism of the translocations and when they occur. Biochemical analyses on the mechanisms of DNA breakage and rejoining permit formulation of detailed models of the human chromosomal translocation process in lymphoid neoplasms. Most human lymphomas are derived from B cells in which a DNA break at an oncogene is initiated by activation-induced deaminase (AID). The partner locus in many cases is located at one of the antigen receptor loci, and this break is generated by the recombination activating gene (RAG) complex or by AID. After breakage, the joining process typically occurs by non-homologous DNA end-joining (NHEJ). Some of the insights into this mechanism also apply to translocations that occur in non-lymphoid neoplasms. |*Translocation, Genetic [MESH] |Animals [MESH] |DNA End-Joining Repair [MESH] |Humans [MESH] |Immunoglobulins/genetics [MESH] |Lymphoma, B-Cell/*genetics [MESH] |Lymphoma, T-Cell/*genetics [MESH]Mechanisms of human lymphoid chromosomal translocations (epmc).pdf DeepDyve Pubget Overpricing