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10.1101/cshperspect.a019372 http://scihub22266oqcxt.onion/10.1101/cshperspect.a019372 C4852805!4852805 !27141050     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27141050 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27141050 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Cold+Spring+Harb+Perspect+Biol 2016 ; 8 (5 ): ä Nephropedia Template TP {{tp|p=27141050 |t=2016. Maintenance of Epigenetic Information |pdf=|usr=}}{{27141050 }} gab.com Text Maintenance of Epigenetic Information JO: Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=27141050 #FOAvip The genome is subject to a diverse array of epigenetic modifications from DNA methylation to histone posttranslational changes. Many of these marks are somatically stable through cell division. This article focuses on our knowledge of the mechanisms governing the inheritance of epigenetic marks, particularly, repressive ones, when the DNA and chromatin template are duplicated in S phase. This involves the action of histone chaperones, nucleosome-remodeling enzymes, histone and DNA methylation binding proteins, and chromatin-modifying enzymes. Last, the timing of DNA replication is discussed, including the question of whether this constitutes an epigenetic mark that facilitates the propagation of epigenetic marks. Twit Text FOAVip Maintenance of Epigenetic Information ????Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=27141050 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27141050 #FOAvip English Wikipedia <ref>{{Cite pmid|27141050 }}</ref> Maintenance of Epigenetic Information #MMPMID27141050 Almouzni G ; Cedar H Cold Spring Harb Perspect Biol 2016[May]; 8 (5 ): ä PMID27141050 show ga The genome is subject to a diverse array of epigenetic modifications from DNA methylation to histone posttranslational changes. Many of these marks are somatically stable through cell division. This article focuses on our knowledge of the mechanisms governing the inheritance of epigenetic marks, particularly, repressive ones, when the DNA and chromatin template are duplicated in S phase. This involves the action of histone chaperones, nucleosome-remodeling enzymes, histone and DNA methylation binding proteins, and chromatin-modifying enzymes. Last, the timing of DNA replication is discussed, including the question of whether this constitutes an epigenetic mark that facilitates the propagation of epigenetic marks. |*Epigenesis, Genetic [MESH] |*Gene Expression Regulation [MESH] |*Models, Genetic [MESH] |DNA Methylation [MESH] |DNA Replication/physiology [MESH] |Histone Chaperones/physiology [MESH] |Histone Code [MESH]Maintenance of Epigenetic Information (epmc).pdf DeepDyve Pubget Overpricing