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http://scihub22266oqcxt.onion/ 19271417!ä!19271417 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=19271417&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\19271417.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Magnes+Res 2008 ; 21 (4): 197-204 Nephropedia Template TP {{tp|p=19271417|t=2008. Magnesium homeostasis and alcohol consumption |pdf=|usr=}}{{19271417}} gab.com Text Magnesium homeostasis and alcohol consumption JO: Magnes Res http://www.kidney.de/pget.php?&tw=1&pmid=19271417 #FOAvip Clinical and experimental evidence indicates alcohol consumption as one of the major causes of magnesium loss from several tissues. As a result of this loss, serum magnesium tends to decrease while urinary magnesium excretion increases 2-3 fold. Experimental data confirm that chronic consumption of 6% ethanol in the Lieber De-Carli diet for 3 weeks results in a marked decrease in total tissue magnesium content in rats. This decrease affects brain, liver and all skeletal muscle, including heart, to a varying extent. While a full picture of the implications of magnesium loss in these tissues is still lacking, it is becoming progressively clear that magnesium loss affects energy production, protein synthesis, cell cycle, and specific functions in the various organs affected. In addition, as magnesium regulated cytokine production and secretion, especially in macrophages and leukocytes, a major role of magnesium deficiency in alcohol-induced inflammatory processes can be envisioned. Considering all these various aspects together, it becomes apparent that magnesium loss may represent a predisposing factor to the onset of alcohol-induced pathologies including brain stroke, sarcopenia, cardiomyopathy, steatohepatitis and cirrhosis. The present review will attempt to clarify some of the mechanisms by which ethanol impairs magnesium transport and homeostasis in brain, brain vasculature, skeletal muscle, heart and liver cells, as a first step towards more mechanistic studies aimed at relating magnesium loss with the incurrence of short- and long-term ethanol-induced complications in these organs. Twit Text FOAVip Magnesium homeostasis and alcohol consumption ????Magnes Res http://www.kidney.de/pget.php?&tw=1&pmid=19271417 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=19271417 #FOAvip English Wikipedia <ref>{{Cite pmid|19271417}}</ref> Magnesium homeostasis and alcohol consumption #MMPMID19271417 Romani AM Magnes Res 2008[Dec]; 21 (4): 197-204 PMID19271417show ga Clinical and experimental evidence indicates alcohol consumption as one of the major causes of magnesium loss from several tissues. As a result of this loss, serum magnesium tends to decrease while urinary magnesium excretion increases 2-3 fold. Experimental data confirm that chronic consumption of 6% ethanol in the Lieber De-Carli diet for 3 weeks results in a marked decrease in total tissue magnesium content in rats. This decrease affects brain, liver and all skeletal muscle, including heart, to a varying extent. While a full picture of the implications of magnesium loss in these tissues is still lacking, it is becoming progressively clear that magnesium loss affects energy production, protein synthesis, cell cycle, and specific functions in the various organs affected. In addition, as magnesium regulated cytokine production and secretion, especially in macrophages and leukocytes, a major role of magnesium deficiency in alcohol-induced inflammatory processes can be envisioned. Considering all these various aspects together, it becomes apparent that magnesium loss may represent a predisposing factor to the onset of alcohol-induced pathologies including brain stroke, sarcopenia, cardiomyopathy, steatohepatitis and cirrhosis. The present review will attempt to clarify some of the mechanisms by which ethanol impairs magnesium transport and homeostasis in brain, brain vasculature, skeletal muscle, heart and liver cells, as a first step towards more mechanistic studies aimed at relating magnesium loss with the incurrence of short- and long-term ethanol-induced complications in these organs. |*Alcohol Drinking[MESH] |*Homeostasis[MESH] |Animals[MESH] |Brain/metabolism[MESH] |Humans[MESH] |Magnesium/*metabolism[MESH] |Muscle, Skeletal/metabolism[MESH]Magnesium homeostasis and alcohol consumption (epmc).pdf DeepDyve Pubget Overpricing