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10.1101/cshperspect.a014423 http://scihub22266oqcxt.onion/10.1101/cshperspect.a014423 C4031955!4031955 !24890833     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24890833 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24890833 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Cold+Spring+Harb+Perspect+Med 2014 ; 4 (6 ): ä Nephropedia Template TP {{tp|p=24890833 |t=2014. MYC and the control of DNA replication |pdf=|usr=}}{{24890833 }} gab.com Text MYC and the control of DNA replication JO: Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=24890833 #FOAvip The MYC oncogene is a multifunctional protein that is aberrantly expressed in a significant fraction of tumors from diverse tissue origins. Because of its multifunctional nature, it has been difficult to delineate the exact contributions of MYC's diverse roles to tumorigenesis. Here, we review the normal role of MYC in regulating DNA replication as well as its ability to generate DNA replication stress when overexpressed. Finally, we discuss the possible mechanisms by which replication stress induced by aberrant MYC expression could contribute to genomic instability and cancer. Twit Text FOAVip MYC and the control of DNA replication ????Cold Spring Harb Perspect Med http://www.kidney.de/pget.php?&tw=1&pmid=24890833 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24890833 #FOAvip English Wikipedia <ref>{{Cite pmid|24890833 }}</ref> MYC and the control of DNA replication #MMPMID24890833 Dominguez-Sola D ; Gautier J Cold Spring Harb Perspect Med 2014[Jun]; 4 (6 ): ä PMID24890833 show ga The MYC oncogene is a multifunctional protein that is aberrantly expressed in a significant fraction of tumors from diverse tissue origins. Because of its multifunctional nature, it has been difficult to delineate the exact contributions of MYC's diverse roles to tumorigenesis. Here, we review the normal role of MYC in regulating DNA replication as well as its ability to generate DNA replication stress when overexpressed. Finally, we discuss the possible mechanisms by which replication stress induced by aberrant MYC expression could contribute to genomic instability and cancer. |*DNA Replication [MESH] |*Gene Expression Regulation, Neoplastic [MESH] |Biomarkers, Tumor/*genetics/metabolism [MESH] |Cell Transformation, Neoplastic/*genetics/metabolism [MESH] |Humans [MESH] |Neoplasms/*genetics/metabolism [MESH]MYC and the control of DNA replication (epmc).pdf DeepDyve Pubget Overpricing