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10.1155/2015/652035 http://scihub22266oqcxt.onion/10.1155/2015/652035 C4629055!4629055 !26568664     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26568664 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mediators+Inflamm 2015 ; 2015 (ä): 652035 Nephropedia Template TP {{tp|p=26568664 |t=2015. Leukocytes: The Double-Edged Sword in Fibrosis |pdf=|usr=}}{{26568664 }} gab.com Text Leukocytes: The Double-Edged Sword in Fibrosis JO: Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=26568664 #FOAvip Skin tissue scar formation and fibrosis are often characterized by the increased production and deposition of extracellular matrix components, accompanied by the accumulation of a vast number of myofibroblasts. Scaring is strongly associated with inflammation and wound healing to regain tissue integrity in response to skin tissue injury. However, increased and uncontrolled inflammation, repetitive injury, and individual predisposition might lead to fibrosis, a severe disorder resulting in the formation of dense and stiff tissue that loses the physical properties and physiological functions of normal tissue. Fibrosis is an extremely complicated and multistage process in which bone marrow-derived leukocytes act as both pro- and antifibrotic agents, and therefore, few, if any, effective therapies are available for the most severe and lethal forms of fibrosis. Herein, we discuss the current knowledge on the multidimensional impact of leukocytes on the induction of fibrosis, focusing on skin fibrosis. Twit Text FOAVip Leukocytes: The Double-Edged Sword in Fibrosis ????Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=26568664 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26568664 #FOAvip English Wikipedia <ref>{{Cite pmid|26568664 }}</ref> Leukocytes: The Double-Edged Sword in Fibrosis #MMPMID26568664 Kryczka J ; Boncela J Mediators Inflamm 2015[]; 2015 (ä): 652035 PMID26568664 show ga Skin tissue scar formation and fibrosis are often characterized by the increased production and deposition of extracellular matrix components, accompanied by the accumulation of a vast number of myofibroblasts. Scaring is strongly associated with inflammation and wound healing to regain tissue integrity in response to skin tissue injury. However, increased and uncontrolled inflammation, repetitive injury, and individual predisposition might lead to fibrosis, a severe disorder resulting in the formation of dense and stiff tissue that loses the physical properties and physiological functions of normal tissue. Fibrosis is an extremely complicated and multistage process in which bone marrow-derived leukocytes act as both pro- and antifibrotic agents, and therefore, few, if any, effective therapies are available for the most severe and lethal forms of fibrosis. Herein, we discuss the current knowledge on the multidimensional impact of leukocytes on the induction of fibrosis, focusing on skin fibrosis. |Epithelial-Mesenchymal Transition [MESH] |Fibroblasts/physiology [MESH] |Fibrosis [MESH] |Humans [MESH] |Leukocytes/*physiology [MESH] |Lymphocytes/physiology [MESH] |Macrophages/physiology [MESH] |Neutrophils/physiology [MESH] |Skin/*pathology [MESH]Leukocytes: The Double-Edged Sword in Fibrosis (epmc).pdf DeepDyve Pubget Overpricing