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10.1523/JNEUROSCI.1547-16.2016 http://scihub22266oqcxt.onion/10.1523/JNEUROSCI.1547-16.2016 C5148235!5148235 !27807158     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27807158 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27807158 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Neurosci 2016 ; 36 (44 ): 11158-11164 Nephropedia Template TP {{tp|p=27807158 |t=2016. Ketamine: NMDA Receptors and Beyond |pdf=|usr=}}{{27807158 }} gab.com Text Ketamine: NMDA Receptors and Beyond JO: J Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=27807158 #FOAvip Human studies examining the effects of the dissociative anesthetic ketamine as a model for psychosis and as a rapidly acting antidepressant have spurred great interest in understanding ketamine's actions at molecular, cellular, and network levels. Although ketamine has unequivocal uncompetitive inhibitory effects on N-methyl-d-aspartate receptors (NMDARs) and may preferentially alter the function of NMDARs on interneurons, recent work has questioned whether block of NMDARs is critical for its mood enhancing actions. In this viewpoint, we examine the evolving literature on ketamine supporting NMDARs as important triggers for certain psychiatric effects and the possibility that the antidepressant trigger is unrelated to NMDARs. The rapidly evolving story of ketamine offers great hope for untangling and treating the biology of both depressive and psychotic illnesses. Twit Text FOAVip Ketamine: NMDA Receptors and Beyond ????J Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=27807158 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27807158 #FOAvip English Wikipedia <ref>{{Cite pmid|27807158 }}</ref> Ketamine: NMDA Receptors and Beyond #MMPMID27807158 Zorumski CF ; Izumi Y ; Mennerick S J Neurosci 2016[Nov]; 36 (44 ): 11158-11164 PMID27807158 show ga Human studies examining the effects of the dissociative anesthetic ketamine as a model for psychosis and as a rapidly acting antidepressant have spurred great interest in understanding ketamine's actions at molecular, cellular, and network levels. Although ketamine has unequivocal uncompetitive inhibitory effects on N-methyl-d-aspartate receptors (NMDARs) and may preferentially alter the function of NMDARs on interneurons, recent work has questioned whether block of NMDARs is critical for its mood enhancing actions. In this viewpoint, we examine the evolving literature on ketamine supporting NMDARs as important triggers for certain psychiatric effects and the possibility that the antidepressant trigger is unrelated to NMDARs. The rapidly evolving story of ketamine offers great hope for untangling and treating the biology of both depressive and psychotic illnesses. |*Ketamine [MESH] |Animals [MESH] |Brain/drug effects/*metabolism [MESH] |Evidence-Based Medicine [MESH] |Hallucinogens [MESH] |Humans [MESH] |Psychoses, Substance-Induced/etiology/*metabolism [MESH] |Receptors, N-Methyl-D-Aspartate/chemistry/*metabolism [MESH]Ketamine: NMDA Receptors and Beyond (epmc).pdf DeepDyve Pubget Overpricing