Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText.
Kick-your-searchterm to multiple Engines kick-your-query now !>
A dictionary by aggregated review articles of nephrology, medicine and the life sciences
Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

suck abstract from ncbi


10.4161/23723548.2014.968059

http://scihub22266oqcxt.onion/10.4161/23723548.2014.968059
suck pdf from google scholar
C4905229!4905229 !27308397
unlimited free pdf from europmc27308397
    free
PDF from PMC    free
html from PMC    free

suck abstract from ncbi


Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27308397 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117
pmid27308397
      Mol+Cell+Oncol 2015 ; 2 (1 ): e968059
Nephropedia Template TP

gab.com Text

Twit Text FOAVip

Twit Text #

English Wikipedia


  • KRAS-driven ROS promote malignant transformation #MMPMID27308397
  • Suh Y ; Lee SJ
  • Mol Cell Oncol 2015[Jan]; 2 (1 ): e968059 PMID27308397 show ga
  • The mechanism underlying KRAS (Kirsten rat sarcoma viral oncogene homolog)-driven cellular transformation remains unclear because of the complexity of its downstream effectors. Park et al. recently reported that levels of reactive oxygen species (ROS) are increased by KRAS and are responsible for KRAS-driven malignant transformation, and further identified the signaling cascade involved as KRAS/p38/PDPK1/PKC?/p47(phox)/NOX1. These findings provide new insight into the molecular mechanisms governing KRAS-driven malignant transformation.
  • ä


  • DeepDyve
  • Pubget Overpricing
  • suck abstract from ncbi

    Linkout box