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10.1016/j.cmet.2015.09.006 http://scihub22266oqcxt.onion/10.1016/j.cmet.2015.09.006 C4635047!4635047 !26437604     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26437604 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Cell+Metab 2015 ; 22 (5 ): 777-87 Nephropedia Template TP {{tp|p=26437604 |t=2015. Ironing out Ferroportin |pdf=|usr=}}{{26437604 }} gab.com Text Ironing out Ferroportin JO: Cell Metab http://www.kidney.de/pget.php?&tw=1&pmid=26437604 #FOAvip Maintaining physiologic iron concentrations in tissues is critical for metabolism and host defense. Iron absorption in the duodenum, recycling of iron from senescent erythrocytes, and iron mobilization from storage in macrophages and hepatocytes constitute the major iron flows into plasma for distribution to tissues, predominantly for erythropoiesis. All iron transfer to plasma occurs through the iron exporter ferroportin. The concentration of functional membrane-associated ferroportin is controlled by its ligand, the iron-regulatory hormone hepcidin, and fine-tuned by regulatory mechanisms serving iron homeostasis, oxygen utilization, host defense, and erythropoiesis. Fundamental questions about the structure and biology of ferroportin remain to be answered. Twit Text FOAVip Ironing out Ferroportin ????Cell Metab http://www.kidney.de/pget.php?&tw=1&pmid=26437604 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26437604 #FOAvip English Wikipedia <ref>{{Cite pmid|26437604 }}</ref> Ironing out Ferroportin #MMPMID26437604 Drakesmith H ; Nemeth E ; Ganz T Cell Metab 2015[Nov]; 22 (5 ): 777-87 PMID26437604 show ga Maintaining physiologic iron concentrations in tissues is critical for metabolism and host defense. Iron absorption in the duodenum, recycling of iron from senescent erythrocytes, and iron mobilization from storage in macrophages and hepatocytes constitute the major iron flows into plasma for distribution to tissues, predominantly for erythropoiesis. All iron transfer to plasma occurs through the iron exporter ferroportin. The concentration of functional membrane-associated ferroportin is controlled by its ligand, the iron-regulatory hormone hepcidin, and fine-tuned by regulatory mechanisms serving iron homeostasis, oxygen utilization, host defense, and erythropoiesis. Fundamental questions about the structure and biology of ferroportin remain to be answered. |Animals [MESH] |Cation Transport Proteins/chemistry/genetics/*metabolism [MESH] |Erythrocytes/metabolism [MESH] |Erythropoiesis/*genetics [MESH] |Ferroportin [MESH] |Hepatocytes/metabolism [MESH] |Hepcidins/chemistry/genetics/*metabolism [MESH] |Homeostasis [MESH] |Humans [MESH] |Iron/chemistry/*metabolism [MESH]Ironing out Ferroportin (epmc).pdf DeepDyve Pubget Overpricing