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10.4103/sjg.SJG_297_17 http://scihub22266oqcxt.onion/10.4103/sjg.SJG_297_17 C5848325!5848325 !29451178     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=29451178 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 219.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29451178 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Saudi+J+Gastroenterol 2018 ; 24 (1 ): 5-11 Nephropedia Template TP {{tp|p=29451178 |t=2018. Involvement of epithelial-mesenchymal transition in liver fibrosis |pdf=|usr=}}{{29451178 }} gab.com Text Involvement of epithelial-mesenchymal transition in liver fibrosis JO: Saudi J Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=29451178 #FOAvip Fibrosis of the liver is an inherent wound healing response to chronic liver injury. Regeneration of liver epithelium and restoration of normal liver structure were generally involved in this process. Although the liver has a striking capacity to adapt to damage through tissue repair, excessive accumulation of extracellular matrix during this process often leads to scar tissue formation and subsequent fibrosis. Epithelial to mesenchymal transition (EMT) enables a polarized epithelial cell to undergo multiple changes biochemically and to bear a mesenchymal cell phenotype. EMT plays a critical role in tissue and organ development and embryogenesis. In the liver, it is proposed that epithelial cells can acquire fibroblastic phonotype via EMT and contribute to fibrogenesis. This made EMT a potential target for antifibrotic strategies. Following an original passion, many investigators devote themselves to exploring this mechanism in liver fibrosis. However, as research continues, this hypothesis became highly controversial. The exact contribution of EMT to fibrogenesis was challenged due to the contradictory results from related studies. In this review, we summarized the recent advances regarding EMT in hepatic fibrosis and discussed the potentially involved liver cell types and pathways in order to reach rational and helpful conclusions. Twit Text FOAVip Involvement of epithelial-mesenchymal transition in liver fibrosis ????Saudi J Gastroenterol http://www.kidney.de/pget.php?&tw=1&pmid=29451178 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29451178 #FOAvip English Wikipedia <ref>{{Cite pmid|29451178 }}</ref> Involvement of epithelial-mesenchymal transition in liver fibrosis #MMPMID29451178 Yu K ; Li Q ; Shi G ; Li N Saudi J Gastroenterol 2018[Jan]; 24 (1 ): 5-11 PMID29451178 show ga Fibrosis of the liver is an inherent wound healing response to chronic liver injury. Regeneration of liver epithelium and restoration of normal liver structure were generally involved in this process. Although the liver has a striking capacity to adapt to damage through tissue repair, excessive accumulation of extracellular matrix during this process often leads to scar tissue formation and subsequent fibrosis. Epithelial to mesenchymal transition (EMT) enables a polarized epithelial cell to undergo multiple changes biochemically and to bear a mesenchymal cell phenotype. EMT plays a critical role in tissue and organ development and embryogenesis. In the liver, it is proposed that epithelial cells can acquire fibroblastic phonotype via EMT and contribute to fibrogenesis. This made EMT a potential target for antifibrotic strategies. Following an original passion, many investigators devote themselves to exploring this mechanism in liver fibrosis. However, as research continues, this hypothesis became highly controversial. The exact contribution of EMT to fibrogenesis was challenged due to the contradictory results from related studies. In this review, we summarized the recent advances regarding EMT in hepatic fibrosis and discussed the potentially involved liver cell types and pathways in order to reach rational and helpful conclusions. |*Epithelial-Mesenchymal Transition [MESH] |Animals [MESH] |Hepatocytes/pathology/physiology [MESH] |Humans [MESH] |Liver Cirrhosis/metabolism/*pathology [MESH] |Liver Regeneration [MESH]Involvement of epithelial-mesenchymal transition in liver fibrosis (epmc).pdf DeepDyve Pubget Overpricing