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10.1111/all.13051 http://scihub22266oqcxt.onion/10.1111/all.13051 C5315659!5315659 !27646878     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27646878 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Allergy 2017 ; 72 (3 ): 331-341 Nephropedia Template TP {{tp|p=27646878 |t=2017. Immunopathology alters Th17 cell glucocorticoid sensitivity |pdf=|usr=}}{{27646878 }} gab.com Text Immunopathology alters Th17 cell glucocorticoid sensitivity JO: Allergy http://www.kidney.de/pget.php?&tw=1&pmid=27646878 #FOAvip Th17 cells contribute to several inflammatory conditions and increasing evidence supports that Th17 cells are glucocorticoid resistant. However, Th17 cells in psoriasis and related diseases are glucocorticoid sensitive. We compare glucocorticoid sensitive and resistant immunological diseases and suggest that several aspects in Th17-related diseases alter glucocorticoid sensitivity of Th17 cells. We identify molecular pathways that are implicated in glucocorticoid sensitivity of Th17 cells in the literature, as this information is useful for developing approaches to overcome glucocorticoid-resistant immunopathology. Twit Text FOAVip Immunopathology alters Th17 cell glucocorticoid sensitivity ????Allergy http://www.kidney.de/pget.php?&tw=1&pmid=27646878 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27646878 #FOAvip English Wikipedia <ref>{{Cite pmid|27646878 }}</ref> Immunopathology alters Th17 cell glucocorticoid sensitivity #MMPMID27646878 Banuelos J ; Cao Y ; Shin SC ; Lu NZ Allergy 2017[Mar]; 72 (3 ): 331-341 PMID27646878 show ga Th17 cells contribute to several inflammatory conditions and increasing evidence supports that Th17 cells are glucocorticoid resistant. However, Th17 cells in psoriasis and related diseases are glucocorticoid sensitive. We compare glucocorticoid sensitive and resistant immunological diseases and suggest that several aspects in Th17-related diseases alter glucocorticoid sensitivity of Th17 cells. We identify molecular pathways that are implicated in glucocorticoid sensitivity of Th17 cells in the literature, as this information is useful for developing approaches to overcome glucocorticoid-resistant immunopathology. |*Drug Tolerance [MESH] |Animals [MESH] |Anti-Inflammatory Agents/metabolism/*pharmacology [MESH] |Cell Differentiation/immunology [MESH] |Cytokines/metabolism [MESH] |Disease Susceptibility [MESH] |Gene Expression Regulation [MESH] |Glucocorticoids/*pharmacology/therapeutic use [MESH] |Humans [MESH] |Immune System Diseases/drug therapy/genetics/immunology/metabolism [MESH] |Inflammation/drug therapy/genetics/immunology/metabolism [MESH] |Signal Transduction [MESH] |T-Lymphocyte Subsets/drug effects/immunology/metabolism [MESH]Immunopathology alters Th17 cell glucocorticoid sensitivity (epmc).pdf DeepDyve Pubget Overpricing