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10.1152/ajprenal.00358.2015 http://scihub22266oqcxt.onion/10.1152/ajprenal.00358.2015 C4967160!4967160 !27009339     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27009339 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Am+J+Physiol+Renal+Physiol 2016 ; 311 (1 ): F16-27 Nephropedia Template TP {{tp|p=27009339 |t=2016. Hypertension: the missing WNKs |pdf=|usr=}}{{27009339 }} gab.com Text Hypertension: the missing WNKs JO: Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27009339 #FOAvip The With no Lysine [K] (WNK) family of enzymes are central in the regulation of blood pressure. WNKs have been implicated in hereditary hypertension disorders, mainly through control of the activity and levels of ion cotransporters and channels. Actions of WNKs in the kidney have been heavily investigated, and recent studies have provided insight into not only the regulation of these enzymes but also how mutations in WNKs and their interacting partners contribute to hypertensive disorders. Defining the roles of WNKs in the cardiovascular system will provide clues about additional mechanisms by which WNKs can regulate blood pressure. This review summarizes recent developments in the regulation of the WNK signaling cascade and its role in regulation of blood pressure. Twit Text FOAVip Hypertension: the missing WNKs ????Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27009339 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27009339 #FOAvip English Wikipedia <ref>{{Cite pmid|27009339 }}</ref> Hypertension: the missing WNKs #MMPMID27009339 Dbouk HA ; Huang CL ; Cobb MH Am J Physiol Renal Physiol 2016[Jul]; 311 (1 ): F16-27 PMID27009339 show ga The With no Lysine [K] (WNK) family of enzymes are central in the regulation of blood pressure. WNKs have been implicated in hereditary hypertension disorders, mainly through control of the activity and levels of ion cotransporters and channels. Actions of WNKs in the kidney have been heavily investigated, and recent studies have provided insight into not only the regulation of these enzymes but also how mutations in WNKs and their interacting partners contribute to hypertensive disorders. Defining the roles of WNKs in the cardiovascular system will provide clues about additional mechanisms by which WNKs can regulate blood pressure. This review summarizes recent developments in the regulation of the WNK signaling cascade and its role in regulation of blood pressure. |Animals [MESH] |Blood Pressure/genetics/physiology [MESH] |Humans [MESH] |Hypertension/*enzymology/*genetics [MESH] |Intracellular Signaling Peptides and Proteins/*genetics [MESH] |Protein Serine-Threonine Kinases/*genetics [MESH]Hypertension: the missing WNKs (epmc).pdf DeepDyve Pubget Overpricing