Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.ccm.2016.04.016 http://scihub22266oqcxt.onion/10.1016/j.ccm.2016.04.016 C4987538!4987538 !27514600     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27514600 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Clin+Chest+Med 2016 ; 37 (3 ): 557-67 Nephropedia Template TP {{tp|p=27514600 |t=2016. Hyper-IgE Syndromes and the Lung |pdf=|usr=}}{{27514600 }} gab.com Text Hyper-IgE Syndromes and the Lung JO: Clin Chest Med http://www.kidney.de/pget.php?&tw=1&pmid=27514600 #FOAvip Elevated serum IgE has many etiologies including parasitic infection, allergy and asthma, malignancy, and immune dysregulation. The hyper-IgE syndromes caused by mutations in STAT3, DOCK8, and PGM3 are monogenic primary immunodeficiencies associated with high IgE, eczema, and recurrent infections. These primary immunodeficiencies are associated with recurrent pneumonias leading to bronchiectasis; however, each has unique features and genetic diagnosis is essential in guiding therapy, discussing family planning, and defining prognosis. This article discusses the clinical features of these primary immunodeficiencies with a particular focus on the pulmonary manifestations and discussion of the genetics, pathogenesis, and approaches to therapy. Twit Text FOAVip Hyper-IgE Syndromes and the Lung ????Clin Chest Med http://www.kidney.de/pget.php?&tw=1&pmid=27514600 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27514600 #FOAvip English Wikipedia <ref>{{Cite pmid|27514600 }}</ref> Hyper-IgE Syndromes and the Lung #MMPMID27514600 Freeman AF ; Olivier KN Clin Chest Med 2016[Sep]; 37 (3 ): 557-67 PMID27514600 show ga Elevated serum IgE has many etiologies including parasitic infection, allergy and asthma, malignancy, and immune dysregulation. The hyper-IgE syndromes caused by mutations in STAT3, DOCK8, and PGM3 are monogenic primary immunodeficiencies associated with high IgE, eczema, and recurrent infections. These primary immunodeficiencies are associated with recurrent pneumonias leading to bronchiectasis; however, each has unique features and genetic diagnosis is essential in guiding therapy, discussing family planning, and defining prognosis. This article discusses the clinical features of these primary immunodeficiencies with a particular focus on the pulmonary manifestations and discussion of the genetics, pathogenesis, and approaches to therapy. |Guanine Nucleotide Exchange Factors/*genetics [MESH] |Humans [MESH] |Job Syndrome/*genetics/immunology/therapy [MESH] |Lung Diseases/*genetics/immunology/therapy [MESH] |Mutation [MESH] |Phosphoglucomutase/*genetics [MESH]Hyper-IgE Syndromes and the Lung (epmc).pdf DeepDyve Pubget Overpricing