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10.1111/cts.12145 http://scihub22266oqcxt.onion/10.1111/cts.12145 C5350952!5350952 !24456587     free     free     free Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 231.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24456587 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Clin+Transl+Sci 2014 ; 7 (2 ): 132-6 Nephropedia Template TP {{tp|p=24456587 |t=2014. Homocysteine metabolism in children with idiopathic nephrotic syndrome |pdf=|usr=}}{{24456587 }} gab.com Text Homocysteine metabolism in children with idiopathic nephrotic syndrome JO: Clin Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=24456587 #FOAvip BACKGROUND: Homocysteine metabolism is altered in children with idiopathic nephrotic syndrome. Hyperhomocysteinemia is a risk factor of early atherosclerosis and glomerulosclerosis and may occur at time of first occurrence of idiopathic nephrotic syndrome. METHODS: Thirty children with first episode of idiopathic nephrotic syndrome (FENS) aged 1-16 years along with 30 age- and sex-matched healthy controls were enrolled in this study. Homocysteine and cysteine were measured with HPLC; vitamin B12 and folic acid were measured with electro-chemilumiscence immunoassay. Primary outcome measure was plasma homocysteine level in children with FENS and in controls. Secondary outcome measures were (1) plasma and urine homocysteine and cysteine levels in children with FENS at 12 weeks and 1 year (remission) and (2) plasma and urine levels of vitamin B12 and folic acid in children with FENS, at 12 weeks and 1 year (remission). RESULTS: Plasma homocysteine and cysteine levels were comparable to controls in children with FENS, at 12 weeks and 1-year remission. Plasma levels of vitamin B12 and folic acid were significantly decreased compared to controls in FENS due to increased urinary excretion, which normalize during remission at 12 weeks and 1 year. Urinary homocysteine and cysteine levels were significantly raised in FENS compared to controls and continued to be raised even at 12-week and 1-year remission. CONCLUSION: Homocysteine metabolism is deranged in children with FENS. Renal effects of long-term raised urinary homocysteine levels need to be studied. Twit Text FOAVip Homocysteine metabolism in children with idiopathic nephrotic syndrome ????Clin Transl Sci http://www.kidney.de/pget.php?&tw=1&pmid=24456587 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24456587 #FOAvip English Wikipedia <ref>{{Cite pmid|24456587 }}</ref> Homocysteine metabolism in children with idiopathic nephrotic syndrome #MMPMID24456587 Kundal M ; Saha A ; Dubey NK ; Kapoor K ; Basak T ; Bhardwaj G ; Tanwar VS ; Sengupta S ; Batra V ; Upadhayay AD ; Bhatt A Clin Transl Sci 2014[Apr]; 7 (2 ): 132-6 PMID24456587 show ga BACKGROUND: Homocysteine metabolism is altered in children with idiopathic nephrotic syndrome. Hyperhomocysteinemia is a risk factor of early atherosclerosis and glomerulosclerosis and may occur at time of first occurrence of idiopathic nephrotic syndrome. METHODS: Thirty children with first episode of idiopathic nephrotic syndrome (FENS) aged 1-16 years along with 30 age- and sex-matched healthy controls were enrolled in this study. Homocysteine and cysteine were measured with HPLC; vitamin B12 and folic acid were measured with electro-chemilumiscence immunoassay. Primary outcome measure was plasma homocysteine level in children with FENS and in controls. Secondary outcome measures were (1) plasma and urine homocysteine and cysteine levels in children with FENS at 12 weeks and 1 year (remission) and (2) plasma and urine levels of vitamin B12 and folic acid in children with FENS, at 12 weeks and 1 year (remission). RESULTS: Plasma homocysteine and cysteine levels were comparable to controls in children with FENS, at 12 weeks and 1-year remission. Plasma levels of vitamin B12 and folic acid were significantly decreased compared to controls in FENS due to increased urinary excretion, which normalize during remission at 12 weeks and 1 year. Urinary homocysteine and cysteine levels were significantly raised in FENS compared to controls and continued to be raised even at 12-week and 1-year remission. CONCLUSION: Homocysteine metabolism is deranged in children with FENS. Renal effects of long-term raised urinary homocysteine levels need to be studied. |Case-Control Studies [MESH] |Child [MESH] |Cholesterol/blood [MESH] |Cysteine/blood/urine [MESH] |Demography [MESH] |Female [MESH] |Folic Acid/blood [MESH] |Homocysteine/blood/*metabolism [MESH] |Humans [MESH] |Male [MESH] |Nephrotic Syndrome/blood/*metabolism/urine [MESH] |Proteinuria/blood [MESH] |Remission Induction [MESH] |Serum Albumin/metabolism [MESH]Homocysteine metabolism in children with idiopathic nephrotic syndrome(epmc).pdf DeepDyve Pubget Overpricing