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10.1101/cshperspect.a018689 http://scihub22266oqcxt.onion/10.1101/cshperspect.a018689 C3970419!3970419 !24691963     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24691963 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Cold+Spring+Harb+Perspect+Biol 2014 ; 6 (4 ): a018689 Nephropedia Template TP {{tp|p=24691963 |t=2014. Histone H3 mutations in pediatric brain tumors |pdf=|usr=}}{{24691963 }} gab.com Text Histone H3 mutations in pediatric brain tumors JO: Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=24691963 #FOAvip Until recently, mutations in histones had not been described in any human disease. However, genome-wide sequencing of pediatric high-grade gliomas revealed somatic heterozygous mutations in the genes encoding histones H3.1 and H3.3, as well as mutations in the chromatin modifiers ATRX and DAXX. The functional significance and mechanistic details of how these mutations affect the tumors is currently under intensive investigation. The information gained from these studies will shed new light on normal brain development as well as increase our understanding of the tumorigenic processes that drive pediatric high-grade gliomas. Twit Text FOAVip Histone H3 mutations in pediatric brain tumors ????Cold Spring Harb Perspect Biol http://www.kidney.de/pget.php?&tw=1&pmid=24691963 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24691963 #FOAvip English Wikipedia <ref>{{Cite pmid|24691963 }}</ref> Histone H3 mutations in pediatric brain tumors #MMPMID24691963 Liu X ; McEachron TA ; Schwartzentruber J ; Wu G Cold Spring Harb Perspect Biol 2014[Apr]; 6 (4 ): a018689 PMID24691963 show ga Until recently, mutations in histones had not been described in any human disease. However, genome-wide sequencing of pediatric high-grade gliomas revealed somatic heterozygous mutations in the genes encoding histones H3.1 and H3.3, as well as mutations in the chromatin modifiers ATRX and DAXX. The functional significance and mechanistic details of how these mutations affect the tumors is currently under intensive investigation. The information gained from these studies will shed new light on normal brain development as well as increase our understanding of the tumorigenic processes that drive pediatric high-grade gliomas. |*Mutation [MESH] |Brain Neoplasms/*genetics [MESH] |Child [MESH] |Epigenomics [MESH] |Glioma/*genetics [MESH] |Histones/*genetics [MESH] |Humans [MESH]Histone H3 mutations in pediatric brain tumors (epmc).pdf DeepDyve Pubget Overpricing