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10.1016/j.tim.2016.01.006 http://scihub22266oqcxt.onion/10.1016/j.tim.2016.01.006 C5319871!5319871 !26875617     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26875617 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26875617 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Microbiol 2016 ; 24 (5 ): 345-355 Nephropedia Template TP {{tp|p=26875617 |t=2016. HIV-1 Reservoirs During Suppressive Therapy |pdf=|usr=}}{{26875617 }} gab.com Text HIV-1 Reservoirs During Suppressive Therapy JO: Trends Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=26875617 #FOAvip The introduction of antiretroviral therapy (ART) 20 years ago has dramatically reduced morbidity and mortality associated with HIV-1. Initially there was hope that ART would be curative, but it quickly became clear that even though ART was able to restore CD4(+) T cell counts and suppress viral loads below levels of detection, discontinuation of treatment resulted in a rapid rebound of infection. This is due to persistence of a small reservoir of latently infected cells with a long half-life, which necessitates life-long ART. Over the past few years, significant progress has been made in defining and characterizing the latent reservoir of HIV-1, and here we review how understanding the latent reservoir during suppressive therapy will lead to significant advances in curative approaches for HIV-1. Twit Text FOAVip HIV-1 Reservoirs During Suppressive Therapy ????Trends Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=26875617 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26875617 #FOAvip English Wikipedia <ref>{{Cite pmid|26875617 }}</ref> HIV-1 Reservoirs During Suppressive Therapy #MMPMID26875617 Barton K ; Winckelmann A ; Palmer S Trends Microbiol 2016[May]; 24 (5 ): 345-355 PMID26875617 show ga The introduction of antiretroviral therapy (ART) 20 years ago has dramatically reduced morbidity and mortality associated with HIV-1. Initially there was hope that ART would be curative, but it quickly became clear that even though ART was able to restore CD4(+) T cell counts and suppress viral loads below levels of detection, discontinuation of treatment resulted in a rapid rebound of infection. This is due to persistence of a small reservoir of latently infected cells with a long half-life, which necessitates life-long ART. Over the past few years, significant progress has been made in defining and characterizing the latent reservoir of HIV-1, and here we review how understanding the latent reservoir during suppressive therapy will lead to significant advances in curative approaches for HIV-1. |Anti-HIV Agents/therapeutic use [MESH] |Anti-Retroviral Agents/*therapeutic use [MESH] |Disease Reservoirs/*virology [MESH] |HIV Infections/blood/*drug therapy/immunology/*virology [MESH] |HIV-1/*drug effects/*physiology [MESH] |Humans [MESH] |Viral Load [MESH]HIV-1 Reservoirs During Suppressive Therapy (epmc).pdf DeepDyve Pubget Overpricing