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http://scihub22266oqcxt.onion/ 26028245!?!26028245 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26028245&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Curr+Hypertens+Rev 2015 ; 11 (1): 49-56 Nephropedia Template TP {{tp|p=26028245|t=2015. Genomics and Pharmacogenomics of Salt-sensitive Hypertension |pdf=|usr=}}{{26028245}} gab.com Text Genomics and Pharmacogenomics of Salt-sensitive Hypertension JO: Curr Hypertens Rev http://www.kidney.de/pget.php?&tw=1&pmid=26028245 #FOAvip Salt sensitivity is estimated to be present in 51% of the hypertensive and 26% of the normotensive populations. The individual blood pressure response to salt is heterogeneous and possibly related to inherited susceptibility. Although the mechanisms underlying salt sensitivity are complex and not well understood, genetics can help to determine the blood response to salt intake. So far only a few genes have been found to be associated with salt-sensitive hypertension using candidate gene association studies. The kidney is critical to overall fluid and electrolyte balance and long-term regulation of blood pressure. Thus, the pathogenesis of salt sensitivity must involve a derangement in renal NaCl handling: an inability to decrease renal sodium transport and increase sodium excretion in the face of an increase in NaCl load that could be caused by aberrant counter-regulatory natriuretic/antinatriuretic pathways. We review here the literature regarding the gene variants associated with salt-sensitive hypertension and how the presence of these gene variants influences the response to antihypertensive therapy. Twit Text FOAVip Genomics and Pharmacogenomics of Salt-sensitive Hypertension ????Curr Hypertens Rev http://www.kidney.de/pget.php?&tw=1&pmid=26028245 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26028245 #FOAvip English Wikipedia <ref>{{Cite pmid|26028245}}</ref> Genomics and Pharmacogenomics of Salt-sensitive Hypertension #MMPMID26028245 Armando I; Villar VA; Jose PA Curr Hypertens Rev 2015[]; 11 (1): 49-56 PMID26028245show ga Salt sensitivity is estimated to be present in 51% of the hypertensive and 26% of the normotensive populations. The individual blood pressure response to salt is heterogeneous and possibly related to inherited susceptibility. Although the mechanisms underlying salt sensitivity are complex and not well understood, genetics can help to determine the blood response to salt intake. So far only a few genes have been found to be associated with salt-sensitive hypertension using candidate gene association studies. The kidney is critical to overall fluid and electrolyte balance and long-term regulation of blood pressure. Thus, the pathogenesis of salt sensitivity must involve a derangement in renal NaCl handling: an inability to decrease renal sodium transport and increase sodium excretion in the face of an increase in NaCl load that could be caused by aberrant counter-regulatory natriuretic/antinatriuretic pathways. We review here the literature regarding the gene variants associated with salt-sensitive hypertension and how the presence of these gene variants influences the response to antihypertensive therapy. |*Genetic Association Studies[MESH] |Blood Pressure/*drug effects[MESH] |Gene Expression Regulation/physiology[MESH] |Genetic Variation[MESH] |Humans[MESH] |Hypertension/*genetics[MESH] |Kidney/*physiopathology[MESH] |Polymorphism, Genetic[MESH] |Reactive Oxygen Species/metabolism[MESH] |Renin-Angiotensin System/drug effects/physiology[MESH]Genomics and Pharmacogenomics of Salt-sensitive Hypertension (epmc).pdf DeepDyve Pubget Overpricing