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10.1038/mp.2014.130 http://scihub22266oqcxt.onion/10.1038/mp.2014.130 C4776744!4776744!25349169     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25349169&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25349169.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mol+Psychiatry 2015 ; 20 (6): 786-92 Nephropedia Template TP {{tp|p=25349169|t=2015. Genetic background of extreme violent behavior |pdf=|usr=}}{{25349169}} gab.com Text Genetic background of extreme violent behavior JO: Mol Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=25349169 #FOAvip In developed countries, the majority of all violent crime is committed by a small group of antisocial recidivistic offenders, but no genes have been shown to contribute to recidivistic violent offending or severe violent behavior, such as homicide. Our results, from two independent cohorts of Finnish prisoners, revealed that a monoamine oxidase A (MAOA) low-activity genotype (contributing to low dopamine turnover rate) as well as the CDH13 gene (coding for neuronal membrane adhesion protein) are associated with extremely violent behavior (at least 10 committed homicides, attempted homicides or batteries). No substantial signal was observed for either MAOA or CDH13 among non-violent offenders, indicating that findings were specific for violent offending, and not largely attributable to substance abuse or antisocial personality disorder. These results indicate both low monoamine metabolism and neuronal membrane dysfunction as plausible factors in the etiology of extreme criminal violent behavior, and imply that at least about 5?10% of all severe violent crime in Finland is attributable to the aforementioned MAOA and CDH13 genotypes. Twit Text FOAVip Genetic background of extreme violent behavior ????Mol Psychiatry http://www.kidney.de/pget.php?&tw=1&pmid=25349169 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25349169 #FOAvip English Wikipedia <ref>{{Cite pmid|25349169}}</ref> Genetic background of extreme violent behavior #MMPMID25349169 Tiihonen J; Rautiainen MR; Ollila H; Repo-Tiihonen E; Virkkunen M; Palotie A; Pietiläinen O; Kristiansson K; Joukamaa M; Lauerma H; Saarela J; Tyni S; Vartiainen H; Paananen J; Goldman D; Paunio T Mol Psychiatry 2015[Jun]; 20 (6): 786-92 PMID25349169show ga In developed countries, the majority of all violent crime is committed by a small group of antisocial recidivistic offenders, but no genes have been shown to contribute to recidivistic violent offending or severe violent behavior, such as homicide. Our results, from two independent cohorts of Finnish prisoners, revealed that a monoamine oxidase A (MAOA) low-activity genotype (contributing to low dopamine turnover rate) as well as the CDH13 gene (coding for neuronal membrane adhesion protein) are associated with extremely violent behavior (at least 10 committed homicides, attempted homicides or batteries). No substantial signal was observed for either MAOA or CDH13 among non-violent offenders, indicating that findings were specific for violent offending, and not largely attributable to substance abuse or antisocial personality disorder. These results indicate both low monoamine metabolism and neuronal membrane dysfunction as plausible factors in the etiology of extreme criminal violent behavior, and imply that at least about 5?10% of all severe violent crime in Finland is attributable to the aforementioned MAOA and CDH13 genotypes. äGenetic background of extreme violent behavior (epmc).pdf DeepDyve Pubget Overpricing