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10.1038/nrc.2016.105 http://scihub22266oqcxt.onion/10.1038/nrc.2016.105 C5279857!5279857 !27782134     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27782134 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27782134 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Nat+Rev+Cancer 2016 ; 16 (12 ): 775-788 Nephropedia Template TP {{tp|p=27782134 |t=2016. Gap junctions and cancer: communicating for 50 years |pdf=|usr=}}{{27782134 }} gab.com Text Gap junctions and cancer: communicating for 50 years JO: Nat Rev Cancer http://www.kidney.de/pget.php?&tw=1&pmid=27782134 #FOAvip Fifty years ago, tumour cells were found to lack electrical coupling, leading to the hypothesis that loss of direct intercellular communication is commonly associated with cancer onset and progression. Subsequent studies linked this phenomenon to gap junctions composed of connexin proteins. Although many studies support the notion that connexins are tumour suppressors, recent evidence suggests that, in some tumour types, they may facilitate specific stages of tumour progression through both junctional and non-junctional signalling pathways. This Timeline article highlights the milestones connecting gap junctions to cancer, and underscores important unanswered questions, controversies and therapeutic opportunities in the field. Twit Text FOAVip Gap junctions and cancer: communicating for 50 years ????Nat Rev Cancer http://www.kidney.de/pget.php?&tw=1&pmid=27782134 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27782134 #FOAvip English Wikipedia <ref>{{Cite pmid|27782134 }}</ref> Gap junctions and cancer: communicating for 50 years #MMPMID27782134 Aasen T ; Mesnil M ; Naus CC ; Lampe PD ; Laird DW Nat Rev Cancer 2016[Dec]; 16 (12 ): 775-788 PMID27782134 show ga Fifty years ago, tumour cells were found to lack electrical coupling, leading to the hypothesis that loss of direct intercellular communication is commonly associated with cancer onset and progression. Subsequent studies linked this phenomenon to gap junctions composed of connexin proteins. Although many studies support the notion that connexins are tumour suppressors, recent evidence suggests that, in some tumour types, they may facilitate specific stages of tumour progression through both junctional and non-junctional signalling pathways. This Timeline article highlights the milestones connecting gap junctions to cancer, and underscores important unanswered questions, controversies and therapeutic opportunities in the field. |*Cell Communication [MESH] |Animals [MESH] |Bystander Effect [MESH] |Connexins/genetics/physiology [MESH] |Gap Junctions/*physiology [MESH] |Genes, Tumor Suppressor [MESH] |Humans [MESH] |Models, Animal [MESH] |Mutation [MESH] |Neoplasms/*physiopathology [MESH]Gap junctions and cancer: communicating for 50 years (epmc).pdf DeepDyve Pubget Overpricing