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10.1007/s12026-015-8705-6 http://scihub22266oqcxt.onion/10.1007/s12026-015-8705-6 C4894527!4894527!26410546     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26410546.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Immunol+Res 2015 ; 63 (1-3): 101-6 Nephropedia Template TP {{tp|p=26410546|t=2015. Fundamental role of C1q in autoimmunity and inflammation |pdf=|usr=}}{{26410546}} gab.com Text Fundamental role of C1q in autoimmunity and inflammation JO: Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=26410546 #FOAvip C1q, historically viewed as the initiating component of the classical complement pathway, also exhibits a variety of complement-independent activities in both innate and acquired immunity. Recent studies focusing on C1q?s suppressive role in the immune system have provided new insight into how abnormal C1q expression and bioactivity may contribute to autoimmunity. In particular, molecular networks involving C1q interactions with cell surface receptors and other ligands are emerging as mechanisms involved in C1q?s modulation of immunity. Here, we discuss the role of C1q in controlling immune cell function, including recently elucidated mechanisms of action, and suggest how these processes are critical for maintaining tissue homeostasis under steady-state conditions and in preventing autoimmunity. Twit Text FOAVip Fundamental role of C1q in autoimmunity and inflammation ????Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=26410546 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26410546 #FOAvip English Wikipedia <ref>{{Cite pmid|26410546}}</ref> Fundamental role of C1q in autoimmunity and inflammation #MMPMID26410546 Son M; Diamond B; Santiago-Schwarz F Immunol Res 2015[Dec]; 63 (1-3): 101-6 PMID26410546show ga C1q, historically viewed as the initiating component of the classical complement pathway, also exhibits a variety of complement-independent activities in both innate and acquired immunity. Recent studies focusing on C1q?s suppressive role in the immune system have provided new insight into how abnormal C1q expression and bioactivity may contribute to autoimmunity. In particular, molecular networks involving C1q interactions with cell surface receptors and other ligands are emerging as mechanisms involved in C1q?s modulation of immunity. Here, we discuss the role of C1q in controlling immune cell function, including recently elucidated mechanisms of action, and suggest how these processes are critical for maintaining tissue homeostasis under steady-state conditions and in preventing autoimmunity. äFundamental role of C1q in autoimmunity and inflammation (epmc).pdf DeepDyve Pubget Overpricing