Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.1016/j.ceb.2014.09.006 http://scihub22266oqcxt.onion/10.1016/j.ceb.2014.09.006 C4252586!4252586 !25269668     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25269668 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Curr+Opin+Cell+Biol 2014 ; 31 (?): 92-7 Nephropedia Template TP {{tp|p=25269668 |t=2014. Forms, forces, and stem cell fate |pdf=|usr=}}{{25269668 }} gab.com Text Forms, forces, and stem cell fate JO: Curr Opin Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=25269668 #FOAvip Cells change their shape and mechanics dramatically during development and tissue healing in response to morphogens, cell-cell contact, adhesion to extracellular matrix, and more. Several regulatory links have been described between cell shape, cytoskeletal tension, matrix adhesiveness and stiffness, and recent studies have begun to uncover how these mechanotransduction pathways can impact transcriptional signaling and cell fate decision. The integrated mechanisms linking cell forces, form and fate are likely critical for driving normal morphogenesis, tissue development, and healing. Dysregulation of these mechanisms may also tip the scale from normal to diseased states. Here, we highlight mechanisms that alter cell shape and mechanics, and the pathways affected by these changes. Twit Text FOAVip Forms, forces, and stem cell fate ????Curr Opin Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=25269668 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25269668 #FOAvip English Wikipedia <ref>{{Cite pmid|25269668 }}</ref> Forms, forces, and stem cell fate #MMPMID25269668 Bellas E ; Chen CS Curr Opin Cell Biol 2014[Dec]; 31 (?): 92-7 PMID25269668 show ga Cells change their shape and mechanics dramatically during development and tissue healing in response to morphogens, cell-cell contact, adhesion to extracellular matrix, and more. Several regulatory links have been described between cell shape, cytoskeletal tension, matrix adhesiveness and stiffness, and recent studies have begun to uncover how these mechanotransduction pathways can impact transcriptional signaling and cell fate decision. The integrated mechanisms linking cell forces, form and fate are likely critical for driving normal morphogenesis, tissue development, and healing. Dysregulation of these mechanisms may also tip the scale from normal to diseased states. Here, we highlight mechanisms that alter cell shape and mechanics, and the pathways affected by these changes. |Animals [MESH] |Cell Differentiation [MESH] |Cell Shape [MESH] |Cytoskeleton/metabolism [MESH] |Extracellular Matrix/metabolism [MESH] |Humans [MESH] |Mechanotransduction, Cellular [MESH] |Morphogenesis [MESH] |Signal Transduction [MESH]Forms, forces, and stem cell fate (epmc).pdf DeepDyve Pubget Overpricing