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10.2147/JIR.S91352 http://scihub22266oqcxt.onion/10.2147/JIR.S91352 C4803250!4803250 !27051312     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27051312 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27051312 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Inflamm+Res 2016 ; 9 (ä): 13-20 Nephropedia Template TP {{tp|p=27051312 |t=2016. Familial Mediterranean fever: current perspectives |pdf=|usr=}}{{27051312 }} gab.com Text Familial Mediterranean fever: current perspectives JO: J Inflamm Res http://www.kidney.de/pget.php?&tw=1&pmid=27051312 #FOAvip Familial Mediterranean fever (FMF) is the most frequent monogenic autoinflammatory disease, and it is characterized by recurrent attacks of fever and polyserositis. The disease is associated with mutations in the MEFV gene encoding pyrin, which causes exaggerated inflammatory response through uncontrolled production of interleukin 1. The major long-term complication of FMF is amyloidosis. Colchicine remains the principle therapy, and the aim of treatment is to prevent acute attacks and the consequences of chronic inflammation. With the evolution in the concepts about the etiopathogenesis and genetics of the disease, we have understood that FMF is more complicated than an ordinary autosomal recessive monogenic disorder. Recently, recommendation sets have been generated for interpretation of genetic testing and genetic diagnosis of FMF. Here, we have reviewed the current perspectives in FMF in light of recent recommendations. Twit Text FOAVip Familial Mediterranean fever: current perspectives ????J Inflamm Res http://www.kidney.de/pget.php?&tw=1&pmid=27051312 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27051312 #FOAvip English Wikipedia <ref>{{Cite pmid|27051312 }}</ref> Familial Mediterranean fever: current perspectives #MMPMID27051312 Sönmez HE ; Batu ED ; Özen S J Inflamm Res 2016[]; 9 (ä): 13-20 PMID27051312 show ga Familial Mediterranean fever (FMF) is the most frequent monogenic autoinflammatory disease, and it is characterized by recurrent attacks of fever and polyserositis. The disease is associated with mutations in the MEFV gene encoding pyrin, which causes exaggerated inflammatory response through uncontrolled production of interleukin 1. The major long-term complication of FMF is amyloidosis. Colchicine remains the principle therapy, and the aim of treatment is to prevent acute attacks and the consequences of chronic inflammation. With the evolution in the concepts about the etiopathogenesis and genetics of the disease, we have understood that FMF is more complicated than an ordinary autosomal recessive monogenic disorder. Recently, recommendation sets have been generated for interpretation of genetic testing and genetic diagnosis of FMF. Here, we have reviewed the current perspectives in FMF in light of recent recommendations. äFamilial Mediterranean fever: current perspectives (epmc).pdf DeepDyve Pubget Overpricing