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10.1016/j.tins.2015.02.001 http://scihub22266oqcxt.onion/10.1016/j.tins.2015.02.001 C4459752!4459752!25765319     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25765319.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Neurosci 2015 ; 38 (4): 237-46 Nephropedia Template TP {{tp|p=25765319|t=2015. Epigenetic Mechanisms of Chronic Pain |pdf=|usr=}}{{25765319}} gab.com Text Epigenetic Mechanisms of Chronic Pain JO: Trends Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25765319 #FOAvip Neuropathic and inflammatory pain promote a large number of persisting adaptations at the cellular and molecular level, allowing tissue or nerve damage, even if only transient, to elicit changes in cells that contribute to the development of chronic pain and associated symptoms. There is evidence that injury-induced changes in chromatin structure drive stable changes in gene expression and neural function, which may cause several symptoms, including allodynia, hyperalgesia, anxiety, and depression. Recent findings on epigenetic changes in the spinal cord and brain during chronic pain may guide fundamental advances in new treatments. In this review, we provide a brief overview of epigenetic regulation in the nervous system and then discuss the still-limited literature that directly implicates epigenetic modifications in chronic pain syndromes. Twit Text FOAVip Epigenetic Mechanisms of Chronic Pain ????Trends Neurosci http://www.kidney.de/pget.php?&tw=1&pmid=25765319 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25765319 #FOAvip English Wikipedia <ref>{{Cite pmid|25765319}}</ref> Epigenetic Mechanisms of Chronic Pain #MMPMID25765319 Descalzi G; Ikegami D; Ushijima T; Nestler E; Zachariou V; Narita M Trends Neurosci 2015[Apr]; 38 (4): 237-46 PMID25765319show ga Neuropathic and inflammatory pain promote a large number of persisting adaptations at the cellular and molecular level, allowing tissue or nerve damage, even if only transient, to elicit changes in cells that contribute to the development of chronic pain and associated symptoms. There is evidence that injury-induced changes in chromatin structure drive stable changes in gene expression and neural function, which may cause several symptoms, including allodynia, hyperalgesia, anxiety, and depression. Recent findings on epigenetic changes in the spinal cord and brain during chronic pain may guide fundamental advances in new treatments. In this review, we provide a brief overview of epigenetic regulation in the nervous system and then discuss the still-limited literature that directly implicates epigenetic modifications in chronic pain syndromes. äEpigenetic Mechanisms of Chronic Pain (epmc).pdf DeepDyve Pubget Overpricing