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10.1042/CS20110506 http://scihub22266oqcxt.onion/10.1042/CS20110506 22248353!ä!22248353 Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=22248353&cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\22248353.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Clin+Sci+(Lond) 2012 ; 122 (9): 397-407 Nephropedia Template TP {{tp|p=22248353|t=2012. Endothelial cells and magnesium: implications in atherosclerosis |pdf=|usr=}}{{22248353}} gab.com Text Endothelial cells and magnesium: implications in atherosclerosis JO: Clin Sci (Lond) http://www.kidney.de/pget.php?&tw=1&pmid=22248353 #FOAvip There is no doubt that the functional and structural integrity of the endothelium is critical in maintaining vascular homoeostasis and in preventing atherosclerosis. In the light of epidemiological and experimental studies, magnesium deficiency is emerging as an inducer of endothelial dysfunction. In particular, data on the effects of low extracellular magnesium on cultured endothelial cells reinforce the idea that correcting magnesium homoeostasis might be a helpful and inexpensive intervention to prevent and treat endothelial dysfunction and, consequently, atherosclerosis. Twit Text FOAVip Endothelial cells and magnesium: implications in atherosclerosis ????Clin Sci (Lond) http://www.kidney.de/pget.php?&tw=1&pmid=22248353 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=22248353 #FOAvip English Wikipedia <ref>{{Cite pmid|22248353}}</ref> Endothelial cells and magnesium: implications in atherosclerosis #MMPMID22248353 Maier JA Clin Sci (Lond) 2012[May]; 122 (9): 397-407 PMID22248353show ga There is no doubt that the functional and structural integrity of the endothelium is critical in maintaining vascular homoeostasis and in preventing atherosclerosis. In the light of epidemiological and experimental studies, magnesium deficiency is emerging as an inducer of endothelial dysfunction. In particular, data on the effects of low extracellular magnesium on cultured endothelial cells reinforce the idea that correcting magnesium homoeostasis might be a helpful and inexpensive intervention to prevent and treat endothelial dysfunction and, consequently, atherosclerosis. |Animals[MESH] |Atherosclerosis/drug therapy/*etiology/*physiopathology[MESH] |Capillary Permeability[MESH] |Endothelial Cells/drug effects/*physiology[MESH] |Humans[MESH] |Interleukin-1alpha/physiology[MESH] |Magnesium Deficiency/*complications/drug therapy/physiopathology[MESH] |Magnesium/pharmacology/*physiology[MESH] |Models, Cardiovascular[MESH] |NF-kappa B/physiology[MESH] |Oxidative Stress[MESH] |Peptide Hydrolases/biosynthesis[MESH]Endothelial cells and magnesium: implications in atherosclerosis (epmc).pdf DeepDyve Pubget Overpricing