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10.1186/s12872-015-0124-z http://scihub22266oqcxt.onion/10.1186/s12872-015-0124-z C4617895!4617895 !26481314     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26481314 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26481314 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       BMC+Cardiovasc+Disord 2015 ; 15 (ä): 130 Nephropedia Template TP {{tp|p=26481314 |t=2015. Endothelial cell control of thrombosis |pdf=|usr=}}{{26481314 }} gab.com Text Endothelial cell control of thrombosis JO: BMC Cardiovasc Disord http://www.kidney.de/pget.php?&tw=1&pmid=26481314 #FOAvip Hemostasis encompasses a set of tightly regulated processes that govern blood clotting, platelet activation, and vascular repair. Upon vascular injury, the hemostatic system initiates a series of vascular events and activates extravascular receptors that act in concert to seal off the damage. Blood clotting is subsequently attenuated by a plethora of inhibitors that prevent excessive clot formation and eventual thrombosis. The endothelium which resides at the interface between the blood and surrounding tissues, serves an integral role in the hemostatic system. Depending on specific tissue needs and local stresses, endothelial cells are capable of evoking either antithrombotic or prothrombotic events. Healthy endothelial cells express antiplatelet and anticoagulant agents that prevent platelet aggregation and fibrin formation, respectively. In the face of endothelial dysfunction, endothelial cells trigger fibrin formation, as well as platelet adhesion and aggregation. Finally, endothelial cells release pro-fibrinolytic agents that initiate fibrinolysis to degrade the clot. Taken together, a functional endothelium is essential to maintain hemostasis and prevent thrombosis. Thus, a greater understanding into the role of the endothelium can provide new avenues for exploration and novel therapies for the management of thromboembolisms. Twit Text FOAVip Endothelial cell control of thrombosis ????BMC Cardiovasc Disord http://www.kidney.de/pget.php?&tw=1&pmid=26481314 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26481314 #FOAvip English Wikipedia <ref>{{Cite pmid|26481314 }}</ref> Endothelial cell control of thrombosis #MMPMID26481314 Yau JW ; Teoh H ; Verma S BMC Cardiovasc Disord 2015[Oct]; 15 (ä): 130 PMID26481314 show ga Hemostasis encompasses a set of tightly regulated processes that govern blood clotting, platelet activation, and vascular repair. Upon vascular injury, the hemostatic system initiates a series of vascular events and activates extravascular receptors that act in concert to seal off the damage. Blood clotting is subsequently attenuated by a plethora of inhibitors that prevent excessive clot formation and eventual thrombosis. The endothelium which resides at the interface between the blood and surrounding tissues, serves an integral role in the hemostatic system. Depending on specific tissue needs and local stresses, endothelial cells are capable of evoking either antithrombotic or prothrombotic events. Healthy endothelial cells express antiplatelet and anticoagulant agents that prevent platelet aggregation and fibrin formation, respectively. In the face of endothelial dysfunction, endothelial cells trigger fibrin formation, as well as platelet adhesion and aggregation. Finally, endothelial cells release pro-fibrinolytic agents that initiate fibrinolysis to degrade the clot. Taken together, a functional endothelium is essential to maintain hemostasis and prevent thrombosis. Thus, a greater understanding into the role of the endothelium can provide new avenues for exploration and novel therapies for the management of thromboembolisms. |Blood Coagulation/physiology [MESH] |Endothelial Cells/*physiology [MESH] |Hemostasis/physiology [MESH] |Humans [MESH] |Neovascularization, Physiologic [MESH] |Platelet Adhesiveness [MESH] |Platelet Aggregation [MESH]Endothelial cell control of thrombosis (epmc).pdf DeepDyve Pubget Overpricing