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10.1155/2016/6813016 http://scihub22266oqcxt.onion/10.1155/2016/6813016 C4829719!4829719!27122657     free     free     free Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 213.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27122657.jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Mediators+Inflamm 2016 ; 2016 (ä): ä Nephropedia Template TP {{tp|p=27122657|t=2016. Endothelial Dysfunction and Inflammation: Immunity in Rheumatoid Arthritis |pdf=|usr=}}{{27122657}} gab.com Text Endothelial Dysfunction and Inflammation: Immunity in Rheumatoid Arthritis JO: Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=27122657 #FOAvip Inflammation, as a feature of rheumatoid arthritis (RA), leads to the activation of endothelial cells (ECs). Activated ECs induce atherosclerosis through an increased expression of leukocyte adhesion molecules. Endothelial dysfunction (ED) is recognized as a failure of endothelial repair mechanisms. It is also an early preclinical marker of atherosclerosis and is commonly found in RA patients. RA is now established as an independent cardiovascular risk factor, while mechanistic determinants of ED in RA are still poorly understood. An expanding body of study has shown that EC at a site of RA is both active participant and regulator of inflammatory process. Over the last decade, a role for endothelial dysfunction in RA associated with cardiovascular disease (CVD) has been hypothesized. At the same time, several maintenance drugs targeting this phenomenon have been tested, which has promising results. Assessment of endothelial function may be a useful tool to identify and monitor RA patients. Twit Text FOAVip Endothelial Dysfunction and Inflammation: Immunity in Rheumatoid Arthritis ????Mediators Inflamm http://www.kidney.de/pget.php?&tw=1&pmid=27122657 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27122657 #FOAvip English Wikipedia <ref>{{Cite pmid|27122657}}</ref> Endothelial Dysfunction and Inflammation: Immunity in Rheumatoid Arthritis #MMPMID27122657 Yang X; Chang Y; Wei W Mediators Inflamm 2016[]; 2016 (ä): ä PMID27122657show ga Inflammation, as a feature of rheumatoid arthritis (RA), leads to the activation of endothelial cells (ECs). Activated ECs induce atherosclerosis through an increased expression of leukocyte adhesion molecules. Endothelial dysfunction (ED) is recognized as a failure of endothelial repair mechanisms. It is also an early preclinical marker of atherosclerosis and is commonly found in RA patients. RA is now established as an independent cardiovascular risk factor, while mechanistic determinants of ED in RA are still poorly understood. An expanding body of study has shown that EC at a site of RA is both active participant and regulator of inflammatory process. Over the last decade, a role for endothelial dysfunction in RA associated with cardiovascular disease (CVD) has been hypothesized. At the same time, several maintenance drugs targeting this phenomenon have been tested, which has promising results. Assessment of endothelial function may be a useful tool to identify and monitor RA patients. äEndothelial Dysfunction and Inflammation: Immunity in Rheumatoid Arthr(epmc).pdf DeepDyve Pubget Overpricing