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10.1161/HYPERTENSIONAHA.116.06599 http://scihub22266oqcxt.onion/10.1161/HYPERTENSIONAHA.116.06599 C4982830!4982830 !27456525     free     free     free Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27456525 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Hypertension 2016 ; 68 (3 ): 526-32 Nephropedia Template TP {{tp|p=27456525 |t=2016. Endogenous Ouabain: Recent Advances and Controversies |pdf=|usr=}}{{27456525 }} gab.com Text Endogenous Ouabain: Recent Advances and Controversies JO: Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=27456525 #FOAvip In this brief article, we summarize recent reports about endogenous ouabain (EO), a cardiotonic steroid (CTS). This includes analysis of mammalian EO, the discovery of EO isomers, regulation of intracellular signaling by EO, and the roles of EO in hypertension, pregnancy, and heart and kidney diseases. Novel ouabain-resistant mice that elucidate the key roles of ?2 Na(+) pumps and their CTS binding site are also discussed. Twit Text FOAVip Endogenous Ouabain: Recent Advances and Controversies ????Hypertension http://www.kidney.de/pget.php?&tw=1&pmid=27456525 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27456525 #FOAvip English Wikipedia <ref>{{Cite pmid|27456525 }}</ref> Endogenous Ouabain: Recent Advances and Controversies #MMPMID27456525 Hamlyn JM ; Blaustein MP Hypertension 2016[Sep]; 68 (3 ): 526-32 PMID27456525 show ga In this brief article, we summarize recent reports about endogenous ouabain (EO), a cardiotonic steroid (CTS). This includes analysis of mammalian EO, the discovery of EO isomers, regulation of intracellular signaling by EO, and the roles of EO in hypertension, pregnancy, and heart and kidney diseases. Novel ouabain-resistant mice that elucidate the key roles of ?2 Na(+) pumps and their CTS binding site are also discussed. |Adrenal Cortex/*metabolism [MESH] |Animals [MESH] |Blood Pressure Determination/methods [MESH] |Cardiovascular Diseases/metabolism/physiopathology [MESH] |Female [MESH] |Humans [MESH] |Hypertension/metabolism/*physiopathology [MESH] |Kidney Diseases/metabolism/physiopathology [MESH] |Male [MESH] |Mice [MESH] |Needs Assessment [MESH] |Ouabain/*metabolism [MESH]Endogenous Ouabain: Recent Advances and Controversies (epmc).pdf DeepDyve Pubget Overpricing