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10.1113/JP273781 http://scihub22266oqcxt.onion/10.1113/JP273781 C5390878!5390878 !28044311     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28044311 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Physiol 2017 ; 595 (8 ): 2431-2437 Nephropedia Template TP {{tp|p=28044311 |t=2017. Effects of hypercapnia on the lung |pdf=|usr=}}{{28044311 }} gab.com Text Effects of hypercapnia on the lung JO: J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=28044311 #FOAvip Gases are sensed by lung cells and can activate specific intracellular signalling pathways, and thus have physiological and pathophysiological effects. Carbon dioxide (CO(2) ), a primary product of oxidative metabolism, can be sensed by eukaryotic cells eliciting specific responses via recently identified signalling pathways. However, the physiological and pathophysiological effects of high CO(2) (hypercapnia) on the lungs and specific lung cells, which are the primary site of CO(2) elimination, are incompletely understood. In this review, we provide a physiological and mechanistic perspective on the effects of hypercapnia on the lungs and discuss the recent understanding of CO(2) modulation of the alveolar epithelial function (lung oedema clearance), epithelial cell repair, innate immunity and airway function. Twit Text FOAVip Effects of hypercapnia on the lung ????J Physiol http://www.kidney.de/pget.php?&tw=1&pmid=28044311 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28044311 #FOAvip English Wikipedia <ref>{{Cite pmid|28044311 }}</ref> Effects of hypercapnia on the lung #MMPMID28044311 Shigemura M ; Lecuona E ; Sznajder JI J Physiol 2017[Apr]; 595 (8 ): 2431-2437 PMID28044311 show ga Gases are sensed by lung cells and can activate specific intracellular signalling pathways, and thus have physiological and pathophysiological effects. Carbon dioxide (CO(2) ), a primary product of oxidative metabolism, can be sensed by eukaryotic cells eliciting specific responses via recently identified signalling pathways. However, the physiological and pathophysiological effects of high CO(2) (hypercapnia) on the lungs and specific lung cells, which are the primary site of CO(2) elimination, are incompletely understood. In this review, we provide a physiological and mechanistic perspective on the effects of hypercapnia on the lungs and discuss the recent understanding of CO(2) modulation of the alveolar epithelial function (lung oedema clearance), epithelial cell repair, innate immunity and airway function. |Animals [MESH] |Carbon Dioxide/metabolism [MESH] |Humans [MESH] |Hypercapnia/*metabolism/pathology [MESH] |Immunity, Innate/*physiology [MESH] |Lung/*metabolism/pathology [MESH]Effects of hypercapnia on the lung (epmc).pdf DeepDyve Pubget Overpricing