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10.1007/s11906-016-0688-8 http://scihub22266oqcxt.onion/10.1007/s11906-016-0688-8 C5083775!5083775 !27787837     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27787837 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 209.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27787837 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Curr+Hypertens+Rep 2016 ; 18 (11 ): 83 Nephropedia Template TP {{tp|p=27787837 |t=2016. Effects of Aspirin on Endothelial Function and Hypertension |pdf=|usr=}}{{27787837 }} gab.com Text Effects of Aspirin on Endothelial Function and Hypertension JO: Curr Hypertens Rep http://www.kidney.de/pget.php?&tw=1&pmid=27787837 #FOAvip PURPOSE OF REVIEW: Endothelial dysfunction is intimately related to the development of various cardiovascular diseases, including hypertension, and is often used as a target for pharmacological treatment. The scope of this review is to assess effects of aspirin on endothelial function and their clinical implication in arterial hypertension. RECENT FINDINGS: Emerging data indicate the role of platelets in the development of vascular inflammation due to the release of proinflammatory mediators, for example, triggered largely by thromboxane. Vascular inflammation further promotes oxidative stress, diminished synthesis of vasodilators, proaggregatory and procoagulant state. These changes translate into vasoconstriction, impaired circulation and thrombotic complications. Aspirin inhibits thromboxane synthesis, abolishes platelets activation and acetylates enzymes switching them to the synthesis of anti-inflammatory substances. Aspirin pleiotropic effects have not been fully elucidated yet. In secondary prevention studies, the decrease in cardiovascular events with aspirin outweighs bleeding risks, but this is not the case in primary prevention settings. Ongoing trials will provide more evidence on whether to expand the use of aspirin or stay within current recommendations. Twit Text FOAVip Effects of Aspirin on Endothelial Function and Hypertension ????Curr Hypertens Rep http://www.kidney.de/pget.php?&tw=1&pmid=27787837 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27787837 #FOAvip English Wikipedia <ref>{{Cite pmid|27787837 }}</ref> Effects of Aspirin on Endothelial Function and Hypertension #MMPMID27787837 Dzeshka MS ; Shantsila A ; Lip GY Curr Hypertens Rep 2016[Nov]; 18 (11 ): 83 PMID27787837 show ga PURPOSE OF REVIEW: Endothelial dysfunction is intimately related to the development of various cardiovascular diseases, including hypertension, and is often used as a target for pharmacological treatment. The scope of this review is to assess effects of aspirin on endothelial function and their clinical implication in arterial hypertension. RECENT FINDINGS: Emerging data indicate the role of platelets in the development of vascular inflammation due to the release of proinflammatory mediators, for example, triggered largely by thromboxane. Vascular inflammation further promotes oxidative stress, diminished synthesis of vasodilators, proaggregatory and procoagulant state. These changes translate into vasoconstriction, impaired circulation and thrombotic complications. Aspirin inhibits thromboxane synthesis, abolishes platelets activation and acetylates enzymes switching them to the synthesis of anti-inflammatory substances. Aspirin pleiotropic effects have not been fully elucidated yet. In secondary prevention studies, the decrease in cardiovascular events with aspirin outweighs bleeding risks, but this is not the case in primary prevention settings. Ongoing trials will provide more evidence on whether to expand the use of aspirin or stay within current recommendations. |*Hypertension/drug therapy [MESH] |Animals [MESH] |Aspirin/*pharmacology [MESH] |Clinical Trials as Topic [MESH] |Endothelium, Vascular/drug effects [MESH] |Humans [MESH] |Thrombosis [MESH]Effects of Aspirin on Endothelial Function and Hypertension (epmc).pdf DeepDyve Pubget Overpricing