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10.1155/2015/420891 http://scihub22266oqcxt.onion/10.1155/2015/420891 C4556818!4556818 !26356073     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=26356073 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 215.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\26356073 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Biomed+Res+Int 2015 ; 2015 (ä): 420891 Nephropedia Template TP {{tp|p=26356073 |t=2015. EMT-Inducing Molecular Factors in Gynecological Cancers |pdf=|usr=}}{{26356073 }} gab.com Text EMT-Inducing Molecular Factors in Gynecological Cancers JO: Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=26356073 #FOAvip Gynecologic cancers are the unregulated growth of neoplastic cells that arise in the cervix, ovaries, fallopian tubes, uterus, vagina, and vulva. Although gynecologic cancers are characterized by different signs and symptoms, studies have shown that they share common risk factors, such as smoking, obesity, age, exposure to certain chemicals, infection with human immunodeficiency virus (HIV), and infection with human papilloma virus (HPV). Despite recent advancements in the preventative, diagnostic, and therapeutic interventions for gynecologic cancers, many patients still die as a result of metastasis and recurrence. Since mounting evidence indicates that the epithelial-mesenchymal transition (EMT) process plays an essential role in metastatic relapse of cancer, understanding the molecular aberrations responsible for the EMT and its underlying signaling should be given high priority in order to reduce cancer morbidity and mortality. Twit Text FOAVip EMT-Inducing Molecular Factors in Gynecological Cancers ????Biomed Res Int http://www.kidney.de/pget.php?&tw=1&pmid=26356073 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=26356073 #FOAvip English Wikipedia <ref>{{Cite pmid|26356073 }}</ref> EMT-Inducing Molecular Factors in Gynecological Cancers #MMPMID26356073 Campo L ; Zhang C ; Breuer EK Biomed Res Int 2015[]; 2015 (ä): 420891 PMID26356073 show ga Gynecologic cancers are the unregulated growth of neoplastic cells that arise in the cervix, ovaries, fallopian tubes, uterus, vagina, and vulva. Although gynecologic cancers are characterized by different signs and symptoms, studies have shown that they share common risk factors, such as smoking, obesity, age, exposure to certain chemicals, infection with human immunodeficiency virus (HIV), and infection with human papilloma virus (HPV). Despite recent advancements in the preventative, diagnostic, and therapeutic interventions for gynecologic cancers, many patients still die as a result of metastasis and recurrence. Since mounting evidence indicates that the epithelial-mesenchymal transition (EMT) process plays an essential role in metastatic relapse of cancer, understanding the molecular aberrations responsible for the EMT and its underlying signaling should be given high priority in order to reduce cancer morbidity and mortality. |Animals [MESH] |Epithelial-Mesenchymal Transition/*physiology [MESH] |Female [MESH] |Genital Neoplasms, Female/*pathology/virology [MESH] |Gynecology/methods [MESH] |HIV/pathogenicity [MESH] |Humans [MESH] |Neoplasm Recurrence, Local/pathology/virology [MESH]EMT-Inducing Molecular Factors in Gynecological Cancers (epmc).pdf DeepDyve Pubget Overpricing