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10.1152/ajprenal.00492.2016 http://scihub22266oqcxt.onion/10.1152/ajprenal.00492.2016 C5407066!5407066 !27927651     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27927651 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27927651 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Am+J+Physiol+Renal+Physiol 2017 ; 312 (4 ): F551-F555 Nephropedia Template TP {{tp|p=27927651 |t=2017. DNA repair in ischemic acute kidney injury |pdf=|usr=}}{{27927651 }} gab.com Text DNA repair in ischemic acute kidney injury JO: Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27927651 #FOAvip Ischemia-reperfusion injury (IRI) is a common cause of acute kidney injury leading to an induction of oxidative stress, cellular dysfunction, and loss of renal function. DNA damage, including oxidative base modifications and physical DNA strand breaks, is a consequence of renal IRI. Like many other organs in the body, a redundant and highly conserved set of endogenous repair pathways have evolved to selectively recognize the various types of cellular DNA damage and combat its negative effects on cell viability. Severe damage to the DNA, however, can trigger cell death and elimination of the injured tubular epithelial cells. In this minireview, we summarize the state of the current field of DNA damage and repair in the kidney and provide some expected and, in some cases, unexpected effects of IRI on DNA damage and repair in the kidney. These findings may be applicable to other forms of acute kidney injury and could provide new opportunities for renal research. Twit Text FOAVip DNA repair in ischemic acute kidney injury ????Am J Physiol Renal Physiol http://www.kidney.de/pget.php?&tw=1&pmid=27927651 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27927651 #FOAvip English Wikipedia <ref>{{Cite pmid|27927651 }}</ref> DNA repair in ischemic acute kidney injury #MMPMID27927651 Pressly JD ; Park F Am J Physiol Renal Physiol 2017[Apr]; 312 (4 ): F551-F555 PMID27927651 show ga Ischemia-reperfusion injury (IRI) is a common cause of acute kidney injury leading to an induction of oxidative stress, cellular dysfunction, and loss of renal function. DNA damage, including oxidative base modifications and physical DNA strand breaks, is a consequence of renal IRI. Like many other organs in the body, a redundant and highly conserved set of endogenous repair pathways have evolved to selectively recognize the various types of cellular DNA damage and combat its negative effects on cell viability. Severe damage to the DNA, however, can trigger cell death and elimination of the injured tubular epithelial cells. In this minireview, we summarize the state of the current field of DNA damage and repair in the kidney and provide some expected and, in some cases, unexpected effects of IRI on DNA damage and repair in the kidney. These findings may be applicable to other forms of acute kidney injury and could provide new opportunities for renal research. |*DNA Damage [MESH] |*DNA Repair [MESH] |Acute Kidney Injury/*genetics/metabolism/pathology [MESH] |Animals [MESH] |Cell Death [MESH] |Cell Proliferation [MESH] |Epithelial Cells/metabolism/pathology [MESH] |Humans [MESH] |Kidney Tubules/*metabolism/pathology [MESH] |Oxidative Stress [MESH] |Reperfusion Injury/*genetics/metabolism/pathology [MESH]DNA repair in ischemic acute kidney injury (epmc).pdf DeepDyve Pubget Overpricing