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2016 ; 18
(2
): 10
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Current Treatments of Bruxism
#MMPMID26897026
Guaita M
; Högl B
Curr Treat Options Neurol
2016[Feb]; 18
(2
): 10
PMID26897026
show ga
Despite numerous case reports, the evidence for treatment of bruxism is still
low. Different treatment modalities (behavioral techniques, intraoral devices,
medications, and contingent electrical stimulation) have been applied. A clinical
evaluation is needed to differentiate between awake bruxism and sleep bruxism and
rule out any medical disorder or medication that could be behind its appearance
(secondary bruxism). A polysomnography is required only in a few cases of sleep
bruxism, mostly when sleep comorbidities are present. Counselling with regard to
sleep hygiene, sleep habit modification, and relaxation techniques has been
suggested as the first step in the therapeutic intervention, and is generally
considered not harmful, despite low evidence of any efficacy. Occlusal splints
are successful in the prevention of dental damage and grinding sounds associated
with sleep bruxism, but their effects on reducing bruxism electromyographic (EMG)
events are transient. In patients with psychiatric and sleep comorbidities, the
acute use of clonazepam at night has been reported to improve sleep bruxism, but
in the absence of double-blind randomized trials, its use in general clinical
practice cannot be recommended. Severe secondary bruxism interfering with
speaking, chewing, or swallowing has been reported in patients with neurological
disorders such as in cranial dystonia; in these patients, injections of botulinum
toxin in the masticatory muscles may decrease bruxism for up to 1-5 months and
improve pain and mandibular functions. Long-term studies in larger and better
specified samples of patients with bruxism, comparing the effects of different
therapeutic modalities on bruxism EMG activity, progression of dental wear, and
orofacial pain are current gaps of knowledge and preclude the development of
severity-based treatment guidelines.