Use my Search Websuite to scan PubMed, PMCentral, Journal Hosts and Journal Archives, FullText. Kick-your-searchterm to multiple Engines kick-your-query now !>

A dictionary by aggregated review articles of nephrology, medicine and the life sciences Your one-stop-run pathway from word to the immediate pdf of peer-reviewed on-topic knowledge.

10.3389/fcimb.2017.00213 http://scihub22266oqcxt.onion/10.3389/fcimb.2017.00213 C5445422!5445422 !28603698     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28603698 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28603698 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Cell+Infect+Microbiol 2017 ; 7 (ä): 213 Nephropedia Template TP {{tp|p=28603698 |t=2017. Crimean-Congo Hemorrhagic Fever: Tick-Host-Virus Interactions |pdf=|usr=}}{{28603698 }} gab.com Text Crimean-Congo Hemorrhagic Fever: Tick-Host-Virus Interactions JO: Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28603698 #FOAvip Crimean-Congo hemorrhagic fever virus (CCHFV) is transmitted to humans by bite of infected ticks or by direct contact with blood or tissues of viremic patients or animals. It causes to humans a severe disease with fatality up to 30%. The current knowledge about the vector-host-CCHFV interactions is very limited due to the high-level containment required for CCHFV studies. Among ticks, Hyalomma spp. are considered the most competent virus vectors. CCHFV evades the tick immune response, and following its replication in the lining of the tick's midgut, it is disseminated by the hemolymph in the salivary glands and reproductive organs. The introduction of salivary gland secretions into the host cells is the major route via which CCHFV enters the host. Following an initial amplification at the site of inoculation, the virus is spread to the target organs. Apoptosis is induced via both intrinsic and extrinsic pathways. Genetic factors and immune status of the host may affect the release of cytokines which play a major role in disease progression and outcome. It is expected that the use of new technology of metabolomics, transcriptomics and proteomics will lead to improved understanding of CCHFV-host interactions and identify potential targets for blocking the CCHFV transmission. Twit Text FOAVip Crimean-Congo Hemorrhagic Fever: Tick-Host-Virus Interactions ????Front Cell Infect Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28603698 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28603698 #FOAvip English Wikipedia <ref>{{Cite pmid|28603698 }}</ref> Crimean-Congo Hemorrhagic Fever: Tick-Host-Virus Interactions #MMPMID28603698 Papa A ; Tsergouli K ; Tsioka K ; Mirazimi A Front Cell Infect Microbiol 2017[]; 7 (ä): 213 PMID28603698 show ga Crimean-Congo hemorrhagic fever virus (CCHFV) is transmitted to humans by bite of infected ticks or by direct contact with blood or tissues of viremic patients or animals. It causes to humans a severe disease with fatality up to 30%. The current knowledge about the vector-host-CCHFV interactions is very limited due to the high-level containment required for CCHFV studies. Among ticks, Hyalomma spp. are considered the most competent virus vectors. CCHFV evades the tick immune response, and following its replication in the lining of the tick's midgut, it is disseminated by the hemolymph in the salivary glands and reproductive organs. The introduction of salivary gland secretions into the host cells is the major route via which CCHFV enters the host. Following an initial amplification at the site of inoculation, the virus is spread to the target organs. Apoptosis is induced via both intrinsic and extrinsic pathways. Genetic factors and immune status of the host may affect the release of cytokines which play a major role in disease progression and outcome. It is expected that the use of new technology of metabolomics, transcriptomics and proteomics will lead to improved understanding of CCHFV-host interactions and identify potential targets for blocking the CCHFV transmission. |Animals [MESH] |Cytokines/metabolism [MESH] |Hemorrhagic Fever Virus, Crimean-Congo/pathogenicity/*physiology [MESH] |Hemorrhagic Fever, Crimean/*transmission [MESH] |Host-Pathogen Interactions/*physiology [MESH] |Humans [MESH] |Ixodidae/immunology/virology [MESH] |Metabolomics/methods [MESH] |Proteomics/methods [MESH] |Salivary Glands/virology [MESH] |Symbiosis [MESH] |Ticks/immunology/*virology [MESH]Crimean-Congo Hemorrhagic Fever: Tick-Host-Virus Interactions (epmc).pdf DeepDyve Pubget Overpricing