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10.1186/s12860-016-0094-8 http://scihub22266oqcxt.onion/10.1186/s12860-016-0094-8 C4896247!4896247 !27229305     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27229305 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\27229305 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       BMC+Cell+Biol 2016 ; 17 Suppl 1 (Suppl 1 ): 12 Nephropedia Template TP {{tp|p=27229305 |t=2016. Connexin and pannexin channels in cancer |pdf=|usr=}}{{27229305 }} gab.com Text Connexin and pannexin channels in cancer JO: BMC Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=27229305 #FOAvip Communication among cells via direct cell-cell contact by connexin gap junctions, or between cell and extracellular environment via pannexin channels or connexin hemichannels, is a key factor in cell function and tissue homeostasis. Upon malignant transformation in different cancer types, the dysregulation of these connexin and pannexin channels and their effect in cellular communication, can either enhance or suppress tumorigenesis and metastasis. In this review, we will highlight the latest reports on the role of the well characterized connexin family and its ability to form gap junctions and hemichannels in cancer. We will also introduce the more recently discovered family of pannexin channels and our current knowledge about their involvement in cancer progression. Twit Text FOAVip Connexin and pannexin channels in cancer ????BMC Cell Biol http://www.kidney.de/pget.php?&tw=1&pmid=27229305 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27229305 #FOAvip English Wikipedia <ref>{{Cite pmid|27229305 }}</ref> Connexin and pannexin channels in cancer #MMPMID27229305 Jiang JX ; Penuela S BMC Cell Biol 2016[May]; 17 Suppl 1 (Suppl 1 ): 12 PMID27229305 show ga Communication among cells via direct cell-cell contact by connexin gap junctions, or between cell and extracellular environment via pannexin channels or connexin hemichannels, is a key factor in cell function and tissue homeostasis. Upon malignant transformation in different cancer types, the dysregulation of these connexin and pannexin channels and their effect in cellular communication, can either enhance or suppress tumorigenesis and metastasis. In this review, we will highlight the latest reports on the role of the well characterized connexin family and its ability to form gap junctions and hemichannels in cancer. We will also introduce the more recently discovered family of pannexin channels and our current knowledge about their involvement in cancer progression. |Animals [MESH] |Cell Cycle [MESH] |Clinical Trials as Topic [MESH] |Connexins/*metabolism [MESH] |Humans [MESH] |Neoplasms/drug therapy/*metabolism/pathology [MESH] |Nerve Tissue Proteins/antagonists & inhibitors/*metabolism [MESH]Connexin and pannexin channels in cancer (epmc).pdf DeepDyve Pubget Overpricing