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10.1016/j.tim.2017.02.003 http://scihub22266oqcxt.onion/10.1016/j.tim.2017.02.003 C5837019!5837019 !28262512     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=28262512 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\28262512 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Trends+Microbiol 2017 ; 25 (5 ): 402-412 Nephropedia Template TP {{tp|p=28262512 |t=2017. Collective Infectious Units in Viruses |pdf=|usr=}}{{28262512 }} gab.com Text Collective Infectious Units in Viruses JO: Trends Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28262512 #FOAvip Increasing evidence indicates that viruses do not simply propagate as independent virions among cells, organs, and hosts. Instead, viral spread is often mediated by structures that simultaneously transport groups of viral genomes, such as polyploid virions, aggregates of virions, virion-containing proteinaceous structures, secreted lipid vesicles, and virus-induced cell-cell contacts. These structures increase the multiplicity of infection, independently of viral population density and transmission bottlenecks. Collective infectious units may contribute to the maintenance of viral genetic diversity, and could have implications for the evolution of social-like virus-virus interactions. These may include various forms of cooperation such as immunity evasion, genetic complementation, division of labor, and relaxation of fitness trade-offs, but also noncooperative interactions such as negative dominance and interference, potentially leading to conflict. Twit Text FOAVip Collective Infectious Units in Viruses ????Trends Microbiol http://www.kidney.de/pget.php?&tw=1&pmid=28262512 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=28262512 #FOAvip English Wikipedia <ref>{{Cite pmid|28262512 }}</ref> Collective Infectious Units in Viruses #MMPMID28262512 Sanjuán R Trends Microbiol 2017[May]; 25 (5 ): 402-412 PMID28262512 show ga Increasing evidence indicates that viruses do not simply propagate as independent virions among cells, organs, and hosts. Instead, viral spread is often mediated by structures that simultaneously transport groups of viral genomes, such as polyploid virions, aggregates of virions, virion-containing proteinaceous structures, secreted lipid vesicles, and virus-induced cell-cell contacts. These structures increase the multiplicity of infection, independently of viral population density and transmission bottlenecks. Collective infectious units may contribute to the maintenance of viral genetic diversity, and could have implications for the evolution of social-like virus-virus interactions. These may include various forms of cooperation such as immunity evasion, genetic complementation, division of labor, and relaxation of fitness trade-offs, but also noncooperative interactions such as negative dominance and interference, potentially leading to conflict. |*Genetic Variation [MESH] |Animals [MESH] |Baculoviridae/genetics/physiology [MESH] |Biological Evolution [MESH] |Virion/*genetics/growth & development/*physiology [MESH]Collective Infectious Units in Viruses (epmc).pdf DeepDyve Pubget Overpricing