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10.1155/2014/149185 http://scihub22266oqcxt.onion/10.1155/2014/149185 C4036716!4036716 !24901008     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=24901008 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\24901008 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       J+Immunol+Res 2014 ; 2014 (ä): 149185 Nephropedia Template TP {{tp|p=24901008 |t=2014. Chronic inflammation and cytokines in the tumor microenvironment |pdf=|usr=}}{{24901008 }} gab.com Text Chronic inflammation and cytokines in the tumor microenvironment JO: J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=24901008 #FOAvip Acute inflammation is a response to an alteration induced by a pathogen or a physical or chemical insult, which functions to eliminate the source of the damage and restore homeostasis to the affected tissue. However, chronic inflammation triggers cellular events that can promote malignant transformation of cells and carcinogenesis. Several inflammatory mediators, such as TNF-?, IL-6, TGF-?, and IL-10, have been shown to participate in both the initiation and progression of cancer. In this review, we explore the role of these cytokines in important events of carcinogenesis, such as their capacity to generate reactive oxygen and nitrogen species, their potential mutagenic effect, and their involvement in mechanisms for epithelial mesenchymal transition, angiogenesis, and metastasis. Finally, we will provide an in-depth analysis of the participation of these cytokines in two types of cancer attributable to chronic inflammatory disease: colitis-associated colorectal cancer and cholangiocarcinoma. Twit Text FOAVip Chronic inflammation and cytokines in the tumor microenvironment ????J Immunol Res http://www.kidney.de/pget.php?&tw=1&pmid=24901008 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=24901008 #FOAvip English Wikipedia <ref>{{Cite pmid|24901008 }}</ref> Chronic inflammation and cytokines in the tumor microenvironment #MMPMID24901008 Landskron G ; De la Fuente M ; Thuwajit P ; Thuwajit C ; Hermoso MA J Immunol Res 2014[]; 2014 (ä): 149185 PMID24901008 show ga Acute inflammation is a response to an alteration induced by a pathogen or a physical or chemical insult, which functions to eliminate the source of the damage and restore homeostasis to the affected tissue. However, chronic inflammation triggers cellular events that can promote malignant transformation of cells and carcinogenesis. Several inflammatory mediators, such as TNF-?, IL-6, TGF-?, and IL-10, have been shown to participate in both the initiation and progression of cancer. In this review, we explore the role of these cytokines in important events of carcinogenesis, such as their capacity to generate reactive oxygen and nitrogen species, their potential mutagenic effect, and their involvement in mechanisms for epithelial mesenchymal transition, angiogenesis, and metastasis. Finally, we will provide an in-depth analysis of the participation of these cytokines in two types of cancer attributable to chronic inflammatory disease: colitis-associated colorectal cancer and cholangiocarcinoma. |Bile Duct Neoplasms/genetics/*metabolism/pathology [MESH] |Bile Ducts, Intrahepatic/metabolism/pathology [MESH] |Carcinogenesis/genetics [MESH] |Cholangiocarcinoma/genetics/*metabolism/pathology [MESH] |Chronic Disease [MESH] |Colitis/complications/genetics/*metabolism/pathology [MESH] |Colorectal Neoplasms/etiology/genetics/*metabolism/pathology [MESH] |Gene Expression [MESH] |Humans [MESH] |Inflammation/complications/genetics/metabolism/pathology [MESH] |Interleukin-10/genetics/metabolism [MESH] |Interleukin-6/genetics/metabolism [MESH] |Oxidative Stress [MESH] |Transforming Growth Factor beta/genetics/metabolism [MESH] |Tumor Microenvironment/*genetics [MESH]Chronic inflammation and cytokines in the tumor microenvironment (epmc).pdf DeepDyve Pubget Overpricing