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10.1002/jnr.23732 http://scihub22266oqcxt.onion/10.1002/jnr.23732 C5415378!5415378 !27117494     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=27117494 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       J+Neurosci+Res 2016 ; 94 (7 ): 609-22 Nephropedia Template TP {{tp|p=27117494 |t=2016. Chronic cerebrovascular dysfunction after traumatic brain injury |pdf=|usr=}}{{27117494 }} gab.com Text Chronic cerebrovascular dysfunction after traumatic brain injury JO: J Neurosci Res http://www.kidney.de/pget.php?&tw=1&pmid=27117494 #FOAvip Traumatic brain injuries (TBI) often involve vascular dysfunction that leads to long-term alterations in physiological and cognitive functions of the brain. Indeed, all the cells that form blood vessels and that are involved in maintaining their proper function can be altered by TBI. This Review focuses on the different types of cerebrovascular dysfunction that occur after TBI, including cerebral blood flow alterations, autoregulation impairments, subarachnoid hemorrhage, vasospasms, blood-brain barrier disruption, and edema formation. We also discuss the mechanisms that mediate these dysfunctions, focusing on the cellular components of cerebral blood vessels (endothelial cells, smooth muscle cells, astrocytes, pericytes, perivascular nerves) and their known and potential roles in the secondary injury cascade. © 2016 Wiley Periodicals, Inc. Twit Text FOAVip Chronic cerebrovascular dysfunction after traumatic brain injury ????J Neurosci Res http://www.kidney.de/pget.php?&tw=1&pmid=27117494 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=27117494 #FOAvip English Wikipedia <ref>{{Cite pmid|27117494 }}</ref> Chronic cerebrovascular dysfunction after traumatic brain injury #MMPMID27117494 Jullienne A ; Obenaus A ; Ichkova A ; Savona-Baron C ; Pearce WJ ; Badaut J J Neurosci Res 2016[Jul]; 94 (7 ): 609-22 PMID27117494 show ga Traumatic brain injuries (TBI) often involve vascular dysfunction that leads to long-term alterations in physiological and cognitive functions of the brain. Indeed, all the cells that form blood vessels and that are involved in maintaining their proper function can be altered by TBI. This Review focuses on the different types of cerebrovascular dysfunction that occur after TBI, including cerebral blood flow alterations, autoregulation impairments, subarachnoid hemorrhage, vasospasms, blood-brain barrier disruption, and edema formation. We also discuss the mechanisms that mediate these dysfunctions, focusing on the cellular components of cerebral blood vessels (endothelial cells, smooth muscle cells, astrocytes, pericytes, perivascular nerves) and their known and potential roles in the secondary injury cascade. © 2016 Wiley Periodicals, Inc. |Animals [MESH] |Blood-Brain Barrier/physiopathology [MESH] |Brain Injuries, Traumatic/*complications/physiopathology [MESH] |Cerebrovascular Circulation [MESH] |Cerebrovascular Disorders/*etiology [MESH] |Chronic Disease [MESH]Chronic cerebrovascular dysfunction after traumatic brain injury (epmc).pdf DeepDyve Pubget Overpricing