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10.1016/j.clp.2014.10.011 http://scihub22266oqcxt.onion/10.1016/j.clp.2014.10.011 C4331454!4331454 !25678002     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=25678002 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215       Clin+Perinatol 2015 ; 42 (1 ): 155-65, ix Nephropedia Template TP {{tp|p=25678002 |t=2015. Chorioamnionitis: implications for the neonate |pdf=|usr=}}{{25678002 }} gab.com Text Chorioamnionitis: implications for the neonate JO: Clin Perinatol http://www.kidney.de/pget.php?&tw=1&pmid=25678002 #FOAvip Chorioamnionitis (CA) is characterized by inflammation of the fetal membranes. The incidence increases with decreasing gestational age at birth. When suspected on clinical criteria, pathologic assessment of the placenta should be performed. Although the mechanisms are not entirely clear, CA predisposes to premature birth, neonatal sepsis, and intraventricular hemorrhage. Its role in respiratory distress syndrome, bronchopulmonary dysplasia, and neurodevelopmental impairment is mixed. Prevention and treatment are ill-defined; antibiotics for preterm premature rupture of membranes reduce the incidence and increase the length of time to delivery. Antibiotics are recommended for infants exposed to CA while laboratory studies are being performed. Twit Text FOAVip Chorioamnionitis: implications for the neonate ????Clin Perinatol http://www.kidney.de/pget.php?&tw=1&pmid=25678002 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25678002 #FOAvip English Wikipedia <ref>{{Cite pmid|25678002 }}</ref> Chorioamnionitis: implications for the neonate #MMPMID25678002 Ericson JE ; Laughon MM Clin Perinatol 2015[Mar]; 42 (1 ): 155-65, ix PMID25678002 show ga Chorioamnionitis (CA) is characterized by inflammation of the fetal membranes. The incidence increases with decreasing gestational age at birth. When suspected on clinical criteria, pathologic assessment of the placenta should be performed. Although the mechanisms are not entirely clear, CA predisposes to premature birth, neonatal sepsis, and intraventricular hemorrhage. Its role in respiratory distress syndrome, bronchopulmonary dysplasia, and neurodevelopmental impairment is mixed. Prevention and treatment are ill-defined; antibiotics for preterm premature rupture of membranes reduce the incidence and increase the length of time to delivery. Antibiotics are recommended for infants exposed to CA while laboratory studies are being performed. |Anti-Bacterial Agents/therapeutic use [MESH] |Bronchopulmonary Dysplasia/epidemiology [MESH] |Cerebral Hemorrhage/*epidemiology [MESH] |Chorioamnionitis/drug therapy/*epidemiology/physiopathology [MESH] |Female [MESH] |Fetal Membranes, Premature Rupture/drug therapy [MESH] |Gestational Age [MESH] |Humans [MESH] |Placenta/pathology [MESH] |Pregnancy [MESH] |Premature Birth/*epidemiology [MESH] |Respiratory Distress Syndrome, Newborn/epidemiology [MESH] |Risk Factors [MESH]Chorioamnionitis: implications for the neonate (epmc).pdf DeepDyve Pubget Overpricing