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10.3389/fphys.2018.00127 http://scihub22266oqcxt.onion/10.3389/fphys.2018.00127 C5829447!5829447 !29527175     free     free     free Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Deprecated: Implicit conversion from float 211.6 to int loses precision in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 534 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\29527175 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Physiol 2018 ; 9 (ä): 127 Nephropedia Template TP {{tp|p=29527175 |t=2018. Cardiac Arrhythmias and Antiarrhythmic Drugs: An Autophagic Perspective |pdf=|usr=}}{{29527175 }} gab.com Text Cardiac Arrhythmias and Antiarrhythmic Drugs: An Autophagic Perspective JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=29527175 #FOAvip Degradation of cellular material by lysosomes is known as autophagy, and its main function is to maintain cellular homeostasis for growth, proliferation and survival of the cell. In recent years, research has focused on the characterization of autophagy pathways. Targeting of autophagy mediators has been described predominantly in cancer treatment, but also in neurological and cardiovascular diseases. Although the number of studies is still limited, there are indications that activity of autophagy pathways increases under arrhythmic conditions. Moreover, an increasing number of antiarrhythmic and non-cardiac drugs are found to affect autophagy pathways. We, therefore, suggest that future work should recognize the largely unaddressed effects of antiarrhythmic agents and other classes of drugs on autophagy pathway activation and inhibition. Twit Text FOAVip Cardiac Arrhythmias and Antiarrhythmic Drugs: An Autophagic Perspective ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=29527175 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=29527175 #FOAvip English Wikipedia <ref>{{Cite pmid|29527175 }}</ref> Cardiac Arrhythmias and Antiarrhythmic Drugs: An Autophagic Perspective #MMPMID29527175 van Bavel JJA ; Vos MA ; van der Heyden MAG Front Physiol 2018[]; 9 (ä): 127 PMID29527175 show ga Degradation of cellular material by lysosomes is known as autophagy, and its main function is to maintain cellular homeostasis for growth, proliferation and survival of the cell. In recent years, research has focused on the characterization of autophagy pathways. Targeting of autophagy mediators has been described predominantly in cancer treatment, but also in neurological and cardiovascular diseases. Although the number of studies is still limited, there are indications that activity of autophagy pathways increases under arrhythmic conditions. Moreover, an increasing number of antiarrhythmic and non-cardiac drugs are found to affect autophagy pathways. We, therefore, suggest that future work should recognize the largely unaddressed effects of antiarrhythmic agents and other classes of drugs on autophagy pathway activation and inhibition. äCardiac Arrhythmias and Antiarrhythmic Drugs: An Autophagic Perspectiv(epmc).pdf DeepDyve Pubget Overpricing