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10.3389/fphys.2020.00747 http://scihub22266oqcxt.onion/10.3389/fphys.2020.00747 C7333313!7333313 !32676039     free     free     free Warning: file_get_contents(https://eutils.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&id=32676039 &cmd=llinks): Failed to open stream: HTTP request failed! HTTP/1.1 429 Too Many Requests in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 215 Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\32676039 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Front+Physiol 2020 ; 11 (ä): 747 Nephropedia Template TP {{tp|p=32676039 |t=2020. COVID-19 Sepsis and Microcirculation Dysfunction |pdf=|usr=}}{{32676039 }} gab.com Text COVID-19 Sepsis and Microcirculation Dysfunction JO: Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=32676039 #FOAvip The spreading of Coronavirus (SARS-CoV-2) pandemic, known as COVID-19, has caused a great number of fatalities all around the World. Up to date (2020 May 6) in Italy we had more than 28,000 deaths, while there were more than 205.000 infected. The majority of patients affected by COVID-19 complained only slight symptoms: fatigue, myalgia or cough, but more than 15% of Chinese patients progressed into severe complications, with acute respiratory distress syndrome (ARDS), needing intensive treatment. We tried to summarize data reported in the last months from several Countries, highlighting that COVID-19 was characterized by cytokine storm (CS) and endothelial dysfunction in severely ill patients, where the progression of the disease was fast and fatal. Endothelial dysfunction was the fundamental mechanism triggering a pro-coagulant state, finally evolving into intravascular disseminated coagulation, causing embolization of several organs and consequent multiorgan failure (MOF). The Italian Society of Clinical Hemorheology and Microcirculation was aimed to highlight the role of microcirculatory dysfunction in the pathogenetic mechanisms of COVID-19 during the spreading of the biggest challenges to the World Health. Twit Text FOAVip COVID-19 Sepsis and Microcirculation Dysfunction ????Front Physiol http://www.kidney.de/pget.php?&tw=1&pmid=32676039 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=32676039 #FOAvip English Wikipedia <ref>{{Cite pmid|32676039 }}</ref> COVID-19 Sepsis and Microcirculation Dysfunction #MMPMID32676039 Colantuoni A ; Martini R ; Caprari P ; Ballestri M ; Capecchi PL ; Gnasso A ; Lo Presti R ; Marcoccia A ; Rossi M ; Caimi G Front Physiol 2020[]; 11 (ä): 747 PMID32676039 show ga The spreading of Coronavirus (SARS-CoV-2) pandemic, known as COVID-19, has caused a great number of fatalities all around the World. Up to date (2020 May 6) in Italy we had more than 28,000 deaths, while there were more than 205.000 infected. The majority of patients affected by COVID-19 complained only slight symptoms: fatigue, myalgia or cough, but more than 15% of Chinese patients progressed into severe complications, with acute respiratory distress syndrome (ARDS), needing intensive treatment. We tried to summarize data reported in the last months from several Countries, highlighting that COVID-19 was characterized by cytokine storm (CS) and endothelial dysfunction in severely ill patients, where the progression of the disease was fast and fatal. Endothelial dysfunction was the fundamental mechanism triggering a pro-coagulant state, finally evolving into intravascular disseminated coagulation, causing embolization of several organs and consequent multiorgan failure (MOF). The Italian Society of Clinical Hemorheology and Microcirculation was aimed to highlight the role of microcirculatory dysfunction in the pathogenetic mechanisms of COVID-19 during the spreading of the biggest challenges to the World Health. äCOVID-19 Sepsis and Microcirculation Dysfunction (epmc).pdf DeepDyve Pubget Overpricing