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10.1152/ajplung.00326.2014 http://scihub22266oqcxt.onion/10.1152/ajplung.00326.2014 C4269691!4269691 !25381027     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25381027 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Am+J+Physiol+Lung+Cell+Mol+Physiol 2014 ; 307 (12 ): L917-23 Nephropedia Template TP {{tp|p=25381027 |t=2014. CFTR and lung homeostasis |pdf=|usr=}}{{25381027 }} gab.com Text CFTR and lung homeostasis JO: Am J Physiol Lung Cell Mol Physiol http://www.kidney.de/pget.php?&tw=1&pmid=25381027 #FOAvip CFTR is a cAMP-activated chloride and bicarbonate channel that is critical for lung homeostasis. Decreases in CFTR expression have dire consequences in cystic fibrosis (CF) and have been suggested to be a component of the lung pathology in chronic obstructive pulmonary disease. Decreases or loss of channel function often lead to mucus stasis, chronic bacterial infections, and the accompanying chronic inflammatory responses that promote progressive lung destruction, and, eventually in CF, lung failure. Here we discuss CFTR's functional role airway surface liquid hydration and pH, in regulation of other channels such as the epithelial sodium channel, and in regulating inflammatory responses in the lung. Twit Text FOAVip CFTR and lung homeostasis ????Am J Physiol Lung Cell Mol Physiol http://www.kidney.de/pget.php?&tw=1&pmid=25381027 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25381027 #FOAvip English Wikipedia <ref>{{Cite pmid|25381027 }}</ref> CFTR and lung homeostasis #MMPMID25381027 Collawn JF ; Matalon S Am J Physiol Lung Cell Mol Physiol 2014[Dec]; 307 (12 ): L917-23 PMID25381027 show ga CFTR is a cAMP-activated chloride and bicarbonate channel that is critical for lung homeostasis. Decreases in CFTR expression have dire consequences in cystic fibrosis (CF) and have been suggested to be a component of the lung pathology in chronic obstructive pulmonary disease. Decreases or loss of channel function often lead to mucus stasis, chronic bacterial infections, and the accompanying chronic inflammatory responses that promote progressive lung destruction, and, eventually in CF, lung failure. Here we discuss CFTR's functional role airway surface liquid hydration and pH, in regulation of other channels such as the epithelial sodium channel, and in regulating inflammatory responses in the lung. |*Homeostasis [MESH] |Animals [MESH] |Cystic Fibrosis Transmembrane Conductance Regulator/genetics/*metabolism [MESH] |Humans [MESH] |Hydrogen-Ion Concentration [MESH] |Lung/*metabolism/pathology [MESH] |Pulmonary Disease, Chronic Obstructive/genetics/*metabolism/pathology [MESH]CFTR and lung homeostasis (epmc).pdf DeepDyve Pubget Overpricing