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10.1186/cc13907 http://scihub22266oqcxt.onion/10.1186/cc13907 C4075125!4075125 !25043644     free     free     free Warning: imagejpeg(C:\Inetpub\vhosts\kidney.de\httpdocs\phplern\25043644 .jpg): Failed to open stream: No such file or directory in C:\Inetpub\vhosts\kidney.de\httpdocs\pget.php on line 117       Crit+Care 2014 ; 18 (3 ): 225 Nephropedia Template TP {{tp|p=25043644 |t=2014. Brain-kidney crosstalk |pdf=|usr=}}{{25043644 }} gab.com Text Brain-kidney crosstalk JO: Crit Care http://www.kidney.de/pget.php?&tw=1&pmid=25043644 #FOAvip Encephalopathy and altered higher mental functions are common clinical complications of acute kidney injury. Although sepsis is a major triggering factor, acute kidney injury predisposes to confusion by causing generalised inflammation, leading to increased permeability of the blood-brain barrier, exacerbated by hyperosmolarity and metabolic acidosis due to the retention of products of nitrogen metabolism potentially resulting in increased brain water content. Downregulation of cell membrane transporters predisposes to alterations in neurotransmitter secretion and uptake, coupled with drug accumulation increasing the risk of encephalopathy. On the other hand, acute brain injury can induce a variety of changes in renal function ranging from altered function and electrolyte imbalances to inflammatory changes in brain death kidney donors. Twit Text FOAVip Brain-kidney crosstalk ????Crit Care http://www.kidney.de/pget.php?&tw=1&pmid=25043644 #FOAvip Twit Text # Free Paper on # # # # # LINK http://www.kidney.de/pget.php?&tw=1&pmid=25043644 #FOAvip English Wikipedia <ref>{{Cite pmid|25043644 }}</ref> Brain-kidney crosstalk #MMPMID25043644 Nongnuch A ; Panorchan K ; Davenport A Crit Care 2014[Jun]; 18 (3 ): 225 PMID25043644 show ga Encephalopathy and altered higher mental functions are common clinical complications of acute kidney injury. Although sepsis is a major triggering factor, acute kidney injury predisposes to confusion by causing generalised inflammation, leading to increased permeability of the blood-brain barrier, exacerbated by hyperosmolarity and metabolic acidosis due to the retention of products of nitrogen metabolism potentially resulting in increased brain water content. Downregulation of cell membrane transporters predisposes to alterations in neurotransmitter secretion and uptake, coupled with drug accumulation increasing the risk of encephalopathy. On the other hand, acute brain injury can induce a variety of changes in renal function ranging from altered function and electrolyte imbalances to inflammatory changes in brain death kidney donors. |Acute Kidney Injury/*complications/metabolism/physiopathology [MESH] |Blood-Brain Barrier/metabolism [MESH] |Brain Death [MESH] |Brain Injuries/*complications/metabolism/physiopathology [MESH] |Confusion/etiology [MESH] |Humans [MESH] |Inflammation/complications [MESH]Brain-kidney crosstalk (epmc).pdf DeepDyve Pubget Overpricing